Inhibition of contractile activity during postconditioning enhances cardioprotection by restoring sarcolemmal dystrophin through phosphatidylinositol 3-kinase

Circulation Journal

Volumn 74, Issue 11, 2010, Pages 2393-2402

  Moriguchi, Akira ^a   Otani, Hajime ^a   Yoshioka, Kei ^a   Shimazu, Takayuki ^a   Fujita, Masanori ^a   Okazaki, Toru ^a   Sato, Daisuke ^a   Kyoi, Shiori ^a   Iwasaka, Toshiji ^a  

^a KANSAI MEDICAL UNIVERSITY   (Japan)

Author keywords

Dystrophin; Phosphatidylinositol 3 kinase; Postconditioning

Indexed keywords

ADENOSINE TRIPHOSPHATE; DIACETYL OXIME; DYSTROPHIN; PHOSPHATIDYLINOSITOL 3 KINASE; PROTEIN KINASE B;

ANIMAL TISSUE; APOPTOSIS; ARTICLE; CELL DEATH; CELLULAR DISTRIBUTION; CONTROLLED STUDY; HEART INFARCTION PREVENTION; HEART INFARCTION SIZE; HEART LEFT VENTRICLE FUNCTION; HEART MUSCLE CELL; HEART MUSCLE CONTRACTILITY; HEART MUSCLE FIBER MEMBRANE; HEART PROTECTION; ISCHEMIC POSTCONDITIONING; ISOLATED HEART; MALE; NONHUMAN; ONCOSIS; PROTEIN PHOSPHORYLATION; RAT;

ADENOSINE TRIPHOSPHATE; ANIMALS; APOPTOSIS; CHROMONES; CYTOPROTECTION; DIACETYL; DYSTROPHIN; ENZYME ACTIVATION; ISCHEMIC POSTCONDITIONING; MALE; MORPHOLINES; MYOCARDIAL CONTRACTION; MYOCARDIAL INFARCTION; MYOCARDIAL REPERFUSION INJURY; MYOCARDIUM; PERFUSION; PHOSPHATIDYLINOSITOL 3-KINASES; PHOSPHORYLATION; PROTEIN KINASE INHIBITORS; PROTEIN TRANSPORT; PROTO-ONCOGENE PROTEINS C-AKT; RATS; RATS, SPRAGUE-DAWLEY; SARCOLEMMA; SIGNAL TRANSDUCTION; TIME FACTORS; VENTRICULAR FUNCTION, LEFT;

EID: 78349249029     PISSN: 13469843     EISSN: 13474820     Source Type: Journal    
DOI: 10.1253/circj.CJ-10-0105     Document Type: Article

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