NA+/H+ exchanger 1- and aquaporin-1-dependent hyperosmolarity changes decrease nitric oxide production and induce VCAM-I expression in endothelial cells exposed to high glucose

International Journal of Immunopathology and Pharmacology

Volumn 23, Issue 3, 2010, Pages 755-765

  Madonna, R ^a   Montebello, E ^a   Lazzerini, G ^b   Zurro, M ^a   De Caterina, R ^a,b  

^a UNIVERSITY OF CHIETI   (Italy)

^b INSTITUTE OF CLINICAL PHYSIOLOGY   (Italy)

Author keywords

Diabetes mellitus; Glucose; Hyperosmolarity; Inflammation; Nitric oxide

Indexed keywords

AQUAPORIN 1; CALPHOSTIN C; CARIPORIDE; DIMETHYL SULFOXIDE; ENDOTHELIAL NITRIC OXIDE SYNTHASE; GLUCOSE; LY 379196; MANNITOL; NITRIC OXIDE; NITRITE; PROTEIN KINASE C BETA; SMALL INTERFERING RNA; SODIUM PROTON EXCHANGE PROTEIN 1; VASCULAR CELL ADHESION MOLECULE 1;

ARTICLE; ENDOTHELIUM CELL; GENE SILENCING; HUMAN; HUMAN CELL; HYPERGLYCEMIA; HYPEROSMOLARITY; IMMUNOBLOTTING; PRIORITY JOURNAL; PROTEIN EXPRESSION;

EID: 78149256392     PISSN: 03946320     EISSN: None     Source Type: Journal    
DOI: 10.1177/039463201002300309     Document Type: Article

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