Notch1 deficiency results in decreased inflammation during wound healing and regulates vascular endothelial growth factor receptor-1 and inflammatory cytokine expression in macrophages

Journal of Immunology

Volumn 185, Issue 7, 2010, Pages 4363-4373

  Outtz, Hasina Hamilton ^a,b   Wu, June K ^b   Wang, Xing ^a,b   Kitajewski, Jan ^a,b  

^a Och Spine at New York Presbyterian Hospitals   (United States)

^b College of Physicians and Surgeons ^*  (United States)

Author keywords

[No Author keywords available]

Indexed keywords

COLLAGEN; CYTOKINE; GAMMA INTERFERON; GAMMA INTERFERON INDUCIBLE PROTEIN 10; INTERLEUKIN 12; INTERLEUKIN 6; LIPOPOLYSACCHARIDE; MONOCYTE CHEMOTACTIC PROTEIN 1; MONOKINE; NOTCH RECEPTOR; NOTCH1 RECEPTOR; TUMOR NECROSIS FACTOR ALPHA; VASCULOTROPIN RECEPTOR 1; NOTCH1 PROTEIN, MOUSE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; BONE MARROW; COLLAGEN DEGRADATION; CONTROLLED STUDY; CYTOKINE PRODUCTION; GENETIC ANALYSIS; GROWTH INHIBITION; IMMUNOHISTOCHEMISTRY; IN VITRO STUDY; INFLAMMATION; MACROPHAGE; MOUSE; MYELOID DENDRITIC CELL; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN INDUCTION; RECEPTOR UPREGULATION; REGULATORY MECHANISM; SIGNAL TRANSDUCTION; VASCULARIZATION; WILD TYPE; WOUND HEALING; ANGIOGENESIS; ANIMAL; BIOSYNTHESIS; CELL SEPARATION; FLOW CYTOMETRY; GENE EXPRESSION; GENE EXPRESSION REGULATION; IMMUNOLOGY; METABOLISM; MOUSE MUTANT; NEUTROPHIL CHEMOTAXIS; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION;

ANIMALS; CELL SEPARATION; CHEMOTAXIS, LEUKOCYTE; CYTOKINES; FLOW CYTOMETRY; GENE EXPRESSION; GENE EXPRESSION REGULATION; IMMUNOHISTOCHEMISTRY; INFLAMMATION; MACROPHAGES; MICE; MICE, MUTANT STRAINS; NEOVASCULARIZATION, PHYSIOLOGIC; RECEPTOR, NOTCH1; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION; SIGNAL TRANSDUCTION; VASCULAR ENDOTHELIAL GROWTH FACTOR RECEPTOR-1; WOUND HEALING;

EID: 77958128428     PISSN: 00221767     EISSN: 15506606     Source Type: Journal    
DOI: 10.4049/jimmunol.1000720     Document Type: Article

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