The 8p11 myeloproliferative syndrome: a distinct clinical entity caused by constitutive activation of FGFR1
Macdonald D., Reiter A., Cross N.C. The 8p11 myeloproliferative syndrome: a distinct clinical entity caused by constitutive activation of FGFR1. Acta Haematol 2002, 107:101-107.
Fusion gene-mediated truncation of RUNX1 as a potential mechanism underlying disease progression in the 8p11 myeloproliferative syndrome
Agerstam H., Lilljebjörn H., Lassen C., Swedin A., Richter J., Vandenberghe P., et al. Fusion gene-mediated truncation of RUNX1 as a potential mechanism underlying disease progression in the 8p11 myeloproliferative syndrome. Genes Chromosomes Cancer 2007, 46:635-643.
Chronic myeloproliferative disorder with t(8;22)(p11;q11) can mime clonal cytogenetic evolution of authentic chronic myelogeneous leukemia
Richebourg S., Theisen O., Plantier I., Parry A., Soenen-Cornu V., Lepelley P., et al. Chronic myeloproliferative disorder with t(8;22)(p11;q11) can mime clonal cytogenetic evolution of authentic chronic myelogeneous leukemia. Genes Chromosomes Cancer 2008, 47:915-918.
The t(8;22) in chronic myeloid leukemia fuses BCR to FGFR1: transforming activity and specific inhibition of FGFR1 fusion proteins
Demiroglu A., Steer E.J., Heath C., Taylor K., Bentley M., Allen S.L., et al. The t(8;22) in chronic myeloid leukemia fuses BCR to FGFR1: transforming activity and specific inhibition of FGFR1 fusion proteins. Blood 2001, 98:3778-3783.
A fourth case of BCR-FGFR1 positive CML-like disease with t(8;22) translocation showing an extensive deletion on the derivative chromosome 8p
Pini M., Gottardi E., Scaravaglio P., Giugliano E., Libener R., Baraldi A., et al. A fourth case of BCR-FGFR1 positive CML-like disease with t(8;22) translocation showing an extensive deletion on the derivative chromosome 8p. Hematol J 2002, 3:315-316.
Fusion of the BCR and the fibroblast growth factor receptor-1 (FGFR1) genes as a result of t(8;22)(p11;q11) in a myeloproliferative disorder: the first fusion gene involving BCR but not ABL
Fioretos T., Panagopoulos I., Lassen C., Swedin A., Billström R., Isaksson M., et al. Fusion of the BCR and the fibroblast growth factor receptor-1 (FGFR1) genes as a result of t(8;22)(p11;q11) in a myeloproliferative disorder: the first fusion gene involving BCR but not ABL. Genes Chromosomes Cancer 2001, 32:302-310.
Dual lympho-myeloproliferative disorder in a patient with t(8;22) with BCR-FGFR1 gene fusion
Murati A., Arnoulet C., Lafage-Pochitaloff M., Adélaide J., Derré M., Slama B., et al. Dual lympho-myeloproliferative disorder in a patient with t(8;22) with BCR-FGFR1 gene fusion. Int J Oncol 2005, 26:1485-1492.
Cooperation of BCR-ABL and AML1/MDS1/EVI1 in blocking myeloid differentiation and rapid induction of an acute myelogenous leukemia
Cuenco G.M., Ren R. Cooperation of BCR-ABL and AML1/MDS1/EVI1 in blocking myeloid differentiation and rapid induction of an acute myelogenous leukemia. Oncogene 2001, 20:8236-8248.
Genome-wide analysis of genetic alterations in acute lymphoblastic leukaemia
Mullighan C.G., Goorha S., Radtke I., Miller C.B., Coustan-Smith E., Dalton J.D., et al. Genome-wide analysis of genetic alterations in acute lymphoblastic leukaemia. Nature 2007, 446:758-764.
Genome-wide analysis of genetic alterations in chronic myelogenous leukemia
(abstract [3363])
Mullighan C.G., Radtke I., Zhang J., Phillips L.A., Su X., Ma J., et al. Genome-wide analysis of genetic alterations in chronic myelogenous leukemia. Blood 2008, 112. (abstract [3363]).