Transient disruption of liver gap junctional intercellular communication and induction of apoptosis after administration of 1,4-bis[2-(3,5 dichloropyridyloxy)]benzene in mice

Experimental and Toxicologic Pathology

Volumn 62, Issue 5, 2010, Pages 525-531

  Fukumasu, Heidge ^a   Sanches, Daniel S ^a   da Silva, Tereza C ^a   Ward, Jerrold M ^b   Dagli, Maria L Z ^a  

^a UNIVERSITY OF SÃO PAULO   (Brazil)

^b Global VetPathology   (United States)

Author keywords

Apoptotic cell death; Cx26; Cx32; Gap junctional intercellular communication; TCPOBOP

Indexed keywords

1,4 BIS(3,5 DICHLORO 2 PYRIDYLOXY)BENZENE; CONNEXIN 26; CONNEXIN 32; CYCLINE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; BODY WEIGHT; CELL COMMUNICATION; CELL PROLIFERATION; CONTROLLED STUDY; FEMALE; GAP JUNCTION; HISTOPATHOLOGY; LIVER TOXICITY; LIVER WEIGHT; MOUSE; NONHUMAN; PROTEIN EXPRESSION;

MUS;

EID: 77955498678     PISSN: 09402993     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.etp.2009.07.001     Document Type: Article

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