Elevated glucose induces congenital heart defects by altering the expression of tbx5, tbx20, and has2 in developing zebrafish embryos

Birth Defects Research Part A - Clinical and Molecular Teratology

Volumn 88, Issue 6, 2010, Pages 480-486

  Liang, Jintao ^a   Gui, Yonghao ^a   Wang, Wei ^a   Gao, Shan ^a   Li, Jing ^a   Song, Houyan ^b  

^a FUDAN UNIVERSITY   (China)

^b FUDAN UNIVERSITY   (China)

Author keywords

Congenital heart defects; Elevated glucose; Growth retardation; has2; tbx20; tbx5; Zebrafish

Indexed keywords

GLUCOSE; LEVO GLUCOSE; TRANSCRIPTION FACTOR; TRANSCRIPTION FACTOR TBX20; TRANSCRIPTION FACTOR TBX5; UNCLASSIFIED DRUG; GLUCURONOSYLTRANSFERASE; HAS2 PROTEIN, ZEBRAFISH; T BOX TRANSCRIPTION FACTOR; T-BOX TRANSCRIPTION FACTOR 5; TBX20 PROTEIN, ZEBRAFISH; ZEBRAFISH PROTEIN;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CONGENITAL HEART MALFORMATION; CONTROLLED STUDY; EMBRYO; GENE; GENE EXPRESSION; GROWTH CURVE; GROWTH RETARDATION; HAS2 GENE; HEART DEVELOPMENT; NEURAL CREST CELL; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; ZEBRA FISH; ANIMAL; EMBRYOLOGY; GENETICS; HEART DEFECTS, CONGENITAL; IN SITU HYBRIDIZATION;

DANIO RERIO;

ANIMALS; GLUCOSE; GLUCURONOSYLTRANSFERASE; HEART DEFECTS, CONGENITAL; IN SITU HYBRIDIZATION; T-BOX DOMAIN PROTEINS; ZEBRAFISH; ZEBRAFISH PROTEINS;

EID: 77953329931     PISSN: 15420752     EISSN: 15420760     Source Type: Journal    
DOI: 10.1002/bdra.20654     Document Type: Article

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