In vitro and in vivo evidence of pathogenic roles of Hic-5/ARA55 in keloids through Smad pathway and profibrotic transcription

Journal of Dermatological Science

Volumn 58, Issue 2, 2010, Pages 152-154

  Inui, Shigeki ^a   Shono, Fumie ^a   Noguchi, Fumihito ^a   Nakajima, Takeshi ^a   Hosokawa, Ko ^a   Itami, Satoshi ^a  

^a OSAKA UNIVERSITY GRADUATE SCHOOL OF MEDICINE   (Japan)

Author keywords

Hic 5; Immunostaining; Keloid; Smad; TGF

Indexed keywords

ANDROGEN RECEPTOR; ANDROGEN RECEPTOR ASSOCIATED PROTEIN 55; COLLAGEN; COLLAGEN TYPE 1; COLLAGEN TYPE 3; GELATINASE A; SMAD PROTEIN; UNCLASSIFIED DRUG;

ADOLESCENT; ADULT; AGED; CLINICAL ARTICLE; COLLAGEN SYNTHESIS; CONTROLLED STUDY; FEMALE; GENE OVEREXPRESSION; HUMAN; HUMAN TISSUE; IMMUNOHISTOCHEMISTRY; IN VITRO STUDY; IN VIVO STUDY; KELOID; LETTER; MALE; PATHOGENESIS; PRIORITY JOURNAL; PROTEIN DEGRADATION; PROTEIN EXPRESSION; PROTEIN FUNCTION; SCAR FORMATION; TRANSCRIPTION REGULATION;

ADULT; AGED; AGED, 80 AND OVER; DNA PRIMERS; FEMALE; FIBROBLASTS; GENE EXPRESSION REGULATION; HUMANS; INTRACELLULAR SIGNALING PEPTIDES AND PROTEINS; KELOID; MALE; MIDDLE AGED; MODELS, BIOLOGICAL; PROTEIN STRUCTURE, TERTIARY; RECEPTORS, TRANSFORMING GROWTH FACTOR BETA; SIGNAL TRANSDUCTION; SKIN; TRANSCRIPTION, GENETIC; TRANSFORMING GROWTH FACTOR BETA;

EID: 77952243840     PISSN: 09231811     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.jdermsci.2010.03.008     Document Type: Letter

References (10)

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