Lack of Mid1, the mouse ortholog of the opitz syndrome gene, causes abnormal development of the anterior cerebellar vermis

Journal of Neuroscience

Volumn 30, Issue 8, 2010, Pages 2880-2887

  Lancioni, Alessio ^a   Pizzo, Mariateresa ^a   Fontanella, Bianca ^a,b   Ferrentino, Rosa ^a   Napolitano, Luisa M R ^a,c   De Leonibus, Elvira ^a   Meroni, Germana ^a,c  

^a TELETHON INSTITUTE OF GENETICS AND MEDICINE TIGEM   (Italy)

^b UNIVERSITY OF SALERNO   (Italy)

^c AREA Science Park   (Italy)

Author keywords

[No Author keywords available]

Indexed keywords

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CEREBELLUM VERMIS; CONTROLLED STUDY; DOWN REGULATION; GENE; IN VIVO STUDY; LEARNING; LEARNING DISORDER; MALE; MESENCEPHALON; MID 1 GENE; MOUSE; NONHUMAN; OPITZ SYNDROME; PATHOGENESIS; PRIORITY JOURNAL; RHOMBENCEPHALON;

ANIMALS; CEREBELLAR CORTEX; CEREBELLAR DISEASES; FEMALE; FIBROBLAST GROWTH FACTORS; GENE EXPRESSION REGULATION, DEVELOPMENTAL; LEARNING DISORDERS; MALE; MESENCEPHALON; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MOVEMENT DISORDERS; NERVOUS SYSTEM MALFORMATIONS; PROTEINS; SYNDROME;

EID: 77649083648     PISSN: 02706474     EISSN: 15292401     Source Type: Journal    
DOI: 10.1523/JNEUROSCI.4196-09.2010     Document Type: Article

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