Arsenite suppression of involucrin transcription through AP1 promoter sites in cultured human keratinocytes

Toxicology and Applied Pharmacology

Volumn 243, Issue 3, 2010, Pages 275-282

  Sinitsyna, Nadezda N ^a   Reznikova, Tatiana V ^a   Qin, Qin ^a   Song, Hyukhwan ^a   Phillips, Marjorie A ^a   Rice, Robert H ^a  

^a UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

c Fos; ChIP assays; Differentiation; Fra 1; Histone H3; Mobility shift

Indexed keywords

ARSENIC TRIOXIDE; CADMIUM; CELL EXTRACT; CHROMIC ACID; HEMIN; HISTONE H3; INVOLUCRIN; PROTEIN C FOS; PROTEIN P300; TRANSCRIPTION FACTOR AP 1; TRANSCRIPTION FACTOR FRA 1; TRANSCRIPTION FACTOR JUNB; VANADIC ACID;

ACETYLATION; ARTICLE; ASSAY; CELL CULTURE; CHROMATIN IMMUNOPRECIPITATION; CONTROLLED STUDY; GEL MOBILITY SHIFT ASSAY; HUMAN; HUMAN CELL; IMMUNOBLOTTING; KERATINOCYTE; PROMOTER REGION; PROTEIN BINDING; REPORTER GENE; TRANSCRIPTION TERMINATION;

ARSENITES; BLOTTING, WESTERN; CELL LINE, TUMOR; CELL NUCLEUS; CELL PROLIFERATION; CELLS, CULTURED; CHROMATIN; ELECTROPHORETIC MOBILITY SHIFT ASSAY; HISTONES; HUMANS; IMMUNOPRECIPITATION; KERATINOCYTES; METALS; P300-CBP TRANSCRIPTION FACTORS; PROMOTER REGIONS, GENETIC; PROTEIN PRECURSORS; RNA, MESSENGER; TRANSCRIPTION FACTOR AP-1; TRANSFECTION;

EID: 77049105614     PISSN: 0041008X     EISSN: 10960333     Source Type: Journal    
DOI: 10.1016/j.taap.2009.12.006     Document Type: Article

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