Deletion of the FHL2 gene attenuates the formation of atherosclerotic lesions after a cholesterol-enriched diet

Life Sciences

Volumn 86, Issue 9-10, 2010, Pages 365-371

  Chu, Pao Hsien ^a   Yeh, Hung I ^b   Wu, Hsueh Hua ^a   Hong, Ray Ching ^b   Shiu, Tzu Fang ^a   Yang, Chuen Mao ^a  

^a CHANG GUNG UNIVERSITY COLLEGE OF MEDICINE   (Taiwan)

^b TAIPEI MEDICAL UNIVERSITY   (Taiwan)

Author keywords

Connexins; Endothelial cells; FHL2; Gap junctions; Hyperlipidemia; Nitrate

Indexed keywords

CHOLESTEROL; CONNEXIN 37; CONNEXIN 40; ENDOTHELIAL NITRIC OXIDE SYNTHASE; FOUR AND A HALF LIM DOMAIN PROTEIN 2; LIM PROTEIN; NITRIC OXIDE; PROTEIN FHL2; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; AORTA; ARTICLE; ATHEROGENESIS; ATHEROSCLEROSIS; ATHEROSCLEROTIC PLAQUE; BLOOD LEVEL; CHOLESTEROL DIET; CONTROLLED STUDY; GAP JUNCTION; GENE EXPRESSION; MALE; MOUSE; MUTANT; NONHUMAN; PROTEIN FAMILY; VASCULAR ENDOTHELIUM; WILD TYPE;

ANIMALS; AORTA; ATHEROSCLEROSIS; CHOLESTEROL, DIETARY; CONNEXINS; ENDOTHELIUM, VASCULAR; GENE DELETION; HOMEODOMAIN PROTEINS; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MUSCLE PROTEINS; NITRIC OXIDE; TRANSCRIPTION FACTORS;

MUS;

EID: 76849085218     PISSN: 00243205     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.lfs.2010.01.007     Document Type: Article

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