Classically and alternatively activated macrophages contribute to tissue remodelling after myocardial infarction

Journal of Cellular and Molecular Medicine

Volumn 13, Issue 9 B, 2009, Pages 3485-3496

  Troidl, C ^a   Mollmann H ^a   Nef, H ^a   Masseli, F ^a   Voss, S ^a   Szardien, S ^a   Willmer, M ^a   Rolf, A ^a   Rixe, J ^a   Troidl, K ^b   Kostin, S ^b   Hamm, C ^b   Elsasser A ^a  

^a KERCKHOFF HEART CENTER   (Germany)

^b MAX PLANCK INSTITUTE FOR HEART AND LUNG RESEARCH   (Germany)

Author keywords

Macrophages; Myocardial infarction; Scar formation; Tissue remodelling

Indexed keywords

ARGINASE; RNA;

ANIMAL; ARTICLE; BIOSYNTHESIS; CYTOLOGY; DNA MICROARRAY; HEART INFARCTION; HEART VENTRICLE REMODELING; IMMUNOHISTOCHEMISTRY; INFLAMMATION; MACROPHAGE; METABOLISM; METHODOLOGY; MONOCYTE; MOUSE; PATHOLOGY; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; SCAR; TIME; UPREGULATION;

ANIMALS; ARGINASE; CICATRIX; IMMUNOHISTOCHEMISTRY; INFLAMMATION; MACROPHAGES; MICE; MONOCYTES; MYOCARDIAL INFARCTION; OLIGONUCLEOTIDE ARRAY SEQUENCE ANALYSIS; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION; RNA; TIME FACTORS; UP-REGULATION; VENTRICULAR REMODELING;

MUS;

EID: 74249095850     PISSN: 15821838     EISSN: None     Source Type: Journal    
DOI: 10.1111/j.1582-4934.2009.00707.x     Document Type: Article

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