Mutant SOD1 G93A microglia have an inflammatory phenotype and elevated production of MCP-1

NeuroReport

Volumn 20, Issue 16, 2009, Pages 1450-1455

  Sargsyan, Siranush A ^a,b   Blackburn, Daniel J ^a   Barber, Siĝn C ^a   Monk, Peter N ^a   Shaw, Pamela J ^a  

^a UNIVERSITY OF SHEFFIELD   (United Kingdom)

^b UNIVERSITY OF COLORADO SCHOOL OF MEDICINE   (United States)

Author keywords

Amyotrophic lateral sclerosis; Microglia; Monocyte chemoattractant protein 1

Indexed keywords

LIPOPOLYSACCHARIDE; MONOCYTE CHEMOTACTIC PROTEIN 1; MUTANT PROTEIN; SUPEROXIDE DISMUTASE; CCL2 PROTEIN, MOUSE; PROSTAGLANDIN E2; SOD1 G93A PROTEIN;

AMYOTROPHIC LATERAL SCLEROSIS; ANIMAL CELL; ARTICLE; CELL ACTIVATION; CELL SURFACE; CONTROLLED STUDY; INFLAMMATION; MICROGLIA; MOUSE; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; PROTEIN DETERMINATION; PROTEIN SYNTHESIS; TRANSGENIC MOUSE; ANIMAL; BRAIN; C57BL MOUSE; CELL CULTURE; CULTURE MEDIUM; CYTOLOGY; DRUG EFFECT; ENZYME LINKED IMMUNOSORBENT ASSAY; GENETICS; HUMAN; METABOLISM; METHODOLOGY; MUTATION; NEWBORN;

ANIMALS; ANIMALS, NEWBORN; BRAIN; CELLS, CULTURED; CHEMOKINE CCL2; CULTURE MEDIA, CONDITIONED; DINOPROSTONE; ENZYME-LINKED IMMUNOSORBENT ASSAY; HUMANS; LIPOPOLYSACCHARIDES; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; MICROGLIA; MUTATION; PHENOTYPE; SUPEROXIDE DISMUTASE;

EID: 74049122793     PISSN: 09594965     EISSN: None     Source Type: Journal    
DOI: 10.1097/WNR.0b013e328331e8fa     Document Type: Article

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