The role of phosphoinositide-3-kinase/Akt pathway in propofol-induced postconditioning against focal cerebral ischemia-reperfusion injury in rats

Brain Research

Volumn 1297, Issue , 2009, Pages 177-184

  Wang, Hai yun ^a   Wang, Guo lin ^a   Yu, Yong hao ^a   Wang, Ying ^a  

^a TIANJIN MEDICAL UNIVERSITY   (China)

Author keywords

Cerebral ischemia reperfusion injury; Phosphoinositide 3 kinase Akt pathway; Postconditioning; Propofol

Indexed keywords

PHOSPHATIDYLINOSITOL 3 KINASE; PROPOFOL; PROTEIN KINASE B; WORTMANNIN;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; BRAIN ISCHEMIA; CONTROLLED STUDY; DRUG EFFICACY; MALE; MIDDLE CEREBRAL ARTERY OCCLUSION; NERVE CELL; NEUROPROTECTION; NICK END LABELING; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; RAT; REPERFUSION INJURY; SIGNAL TRANSDUCTION;

1-PHOSPHATIDYLINOSITOL 3-KINASE; ANDROSTADIENES; ANIMALS; BRAIN; BRAIN INFARCTION; DISEASE MODELS, ANIMAL; DOSE-RESPONSE RELATIONSHIP, DRUG; DRUG ADMINISTRATION SCHEDULE; ENZYME INHIBITORS; HYPNOTICS AND SEDATIVES; HYPOXIA-ISCHEMIA, BRAIN; INFARCTION, MIDDLE CEREBRAL ARTERY; MALE; NEUROPROTECTIVE AGENTS; PHOSPHORYLATION; PROPOFOL; PROTO-ONCOGENE PROTEINS C-AKT; RATS; RATS, SPRAGUE-DAWLEY; REPERFUSION INJURY; SIGNAL TRANSDUCTION; TREATMENT OUTCOME; UP-REGULATION;

RATTUS;

EID: 71749104867     PISSN: 00068993     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.brainres.2009.08.054     Document Type: Article

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