-
1
-
-
0029180674
-
Hepatocellular dysfunction occurs earlier than the onset of hyperdynamic circulation during sepsis
-
Wang P, Ba ZF and Chaudry IH: Hepatocellular dysfunction occurs earlier than the onset of hyperdynamic circulation during sepsis. Shock 3: 21-26, 1995.
-
(1995)
Shock
, vol.3
, pp. 21-26
-
-
Wang, P.1
Ba, Z.F.2
Chaudry, I.H.3
-
2
-
-
0033562174
-
Kupffer cells are responsible for producing inflammatory cytokines and hepatocellular dysfunction during early sepsis
-
Koo DJ, Chaudry IH and Wang P: Kupffer cells are responsible for producing inflammatory cytokines and hepatocellular dysfunction during early sepsis. J Surg Res 83: 151-157, 1999.
-
(1999)
J Surg Res
, vol.83
, pp. 151-157
-
-
Koo, D.J.1
Chaudry, I.H.2
Wang, P.3
-
3
-
-
0036884978
-
Mechanisms of the beneficial effect of adrenomedullin and adrenomedullin-binding protein-1 in sepsis: Down-regulation of proinflammatory cytokines
-
Yang S, Zhou M, Fowler DE and Wang P: Mechanisms of the beneficial effect of adrenomedullin and adrenomedullin-binding protein-1 in sepsis: down-regulation of proinflammatory cytokines. Crit Care Med 30: 2729-2735, 2002.
-
(2002)
Crit Care Med
, vol.30
, pp. 2729-2735
-
-
Yang, S.1
Zhou, M.2
Fowler, D.E.3
Wang, P.4
-
4
-
-
0029382098
-
Sustained elevation in circulating catecholamine levels during polymicrobial sepsis
-
Hahn PY, Wang P, Tait SM, Ba ZF, Reich SS and Chaudry IH: Sustained elevation in circulating catecholamine levels during polymicrobial sepsis. Shock 4: 269-273, 1995.
-
(1995)
Shock
, vol.4
, pp. 269-273
-
-
Hahn, P.Y.1
Wang, P.2
Tait, S.M.3
Ba, Z.F.4
Reich, S.S.5
Chaudry, I.H.6
-
5
-
-
0023084552
-
Plasma catecholamines following cecal ligation and puncture in the rat
-
Kovarik MF, Jones SB and Romano FD: Plasma catecholamines following cecal ligation and puncture in the rat. Circ Shock 22: 281-290, 1987.
-
(1987)
Circ Shock
, vol.22
, pp. 281-290
-
-
Kovarik, M.F.1
Jones, S.B.2
Romano, F.D.3
-
6
-
-
0033635547
-
Gut-derived norepinephrine plays a critical role in producing hepatocellular dysfunction during early sepsis
-
Yang S, Koo DJ, Zhou M, Chaudry IH and Wang P: Gut-derived norepinephrine plays a critical role in producing hepatocellular dysfunction during early sepsis. Am J Physiol Gastrointest Liver Physiol 279: G1274-G1281, 2000.
-
(2000)
Am J Physiol Gastrointest Liver Physiol
, vol.279
-
-
Yang, S.1
Koo, D.J.2
Zhou, M.3
Chaudry, I.H.4
Wang, P.5
-
7
-
-
0028966975
-
Production and metabolism of dopamine and norepinephrine in mesenteric organs and liver of swine
-
Eisenhofer G, Aneman A, Hooper D, Holmes C, Goldstein DS and Friberg P: Production and metabolism of dopamine and norepinephrine in mesenteric organs and liver of swine. Am J Physiol 268: G641-G649, 1995.
-
(1995)
Am J Physiol
, vol.268
-
-
Eisenhofer, G.1
Aneman, A.2
Hooper, D.3
Holmes, C.4
Goldstein, D.S.5
Friberg, P.6
-
8
-
-
0029874309
-
Mesenteric organ production, hepatic metabolism, and renal elimination of norepinephrine and its metabolites in humans
-
Eisenhofer G, Aneman A, Hooper D, Rundqvist B and Friberg P: Mesenteric organ production, hepatic metabolism, and renal elimination of norepinephrine and its metabolites in humans. J Neurochem 66: 1565-1573, 1996.
-
(1996)
J Neurochem
, vol.66
, pp. 1565-1573
-
-
Eisenhofer, G.1
Aneman, A.2
Hooper, D.3
Rundqvist, B.4
Friberg, P.5
-
9
-
-
0034786538
-
Norepinephrine-induced hepatocellular dysfunction in early sepsis is mediated by activation of alpha2-adrenoceptors
-
Yang S, Zhou M, Chaudry IH and Wang P: Norepinephrine-induced hepatocellular dysfunction in early sepsis is mediated by activation of alpha2-adrenoceptors. Am J Physiol Gastrointest Liver Physiol 281: G1014-G1021, 2001.
-
(2001)
Am J Physiol Gastrointest Liver Physiol
, vol.281
-
-
Yang, S.1
Zhou, M.2
Chaudry, I.H.3
Wang, P.4
-
10
-
-
65549157193
-
Pivotal role of the alpha(2A)-adrenoceptor in producing inflammation and organ injury in a rat model of sepsis
-
Miksa M, Das P, Zhou M, et al: Pivotal role of the alpha(2A)-adrenoceptor in producing inflammation and organ injury in a rat model of sepsis. PLoS One 4: E5504, 2009.
-
(2009)
PLoS One
, vol.4
-
-
Miksa, M.1
Das, P.2
Zhou, M.3
-
11
-
-
0025070946
-
Stimulation of alpha-adrenergic receptor augments the production of macrophage-derived tumor necrosis factor
-
Spengler RN, Allen RM, Remick DG, Strieter RM and Kunkel SL: Stimulation of alpha-adrenergic receptor augments the production of macrophage-derived tumor necrosis factor. J Immunol 145: 1430-1434, 1990.
-
(1990)
J Immunol
, vol.145
, pp. 1430-1434
-
-
Spengler, R.N.1
Allen, R.M.2
Remick, D.G.3
Strieter, R.M.4
Kunkel, S.L.5
-
12
-
-
31344442772
-
MAP kinase phosphatase 1 controls innate immune responses and suppresses endotoxic shock
-
Zhao Q, Wang X, Nelin LD, et al: MAP kinase phosphatase 1 controls innate immune responses and suppresses endotoxic shock. J Exp Med 203: 131-140, 2006.
-
(2006)
J Exp Med
, vol.203
, pp. 131-140
-
-
Zhao, Q.1
Wang, X.2
Nelin, L.D.3
-
13
-
-
31344475056
-
Dual specificity phosphatase 1 (DUSP1) regulates a subset of LPS-induced genes and protects mice from lethal endotoxin shock
-
Hammer M, Mages J, Dietrich H, et al: Dual specificity phosphatase 1 (DUSP1) regulates a subset of LPS-induced genes and protects mice from lethal endotoxin shock. J Exp Med 203: 15-20, 2006.
-
(2006)
J Exp Med
, vol.203
, pp. 15-20
-
-
Hammer, M.1
Mages, J.2
Dietrich, H.3
-
14
-
-
31144452241
-
Essential role of MAPK phosphatase-1 in the negative control of innate immune responses
-
Salojin KV, Owusu IB, Millerchip KA, Potter M, Platt KA and Oravecz T: Essential role of MAPK phosphatase-1 in the negative control of innate immune responses. J Immunol 176: 1899-1907, 2006.
-
(2006)
J Immunol
, vol.176
, pp. 1899-1907
-
-
Salojin, K.V.1
Owusu, I.B.2
Millerchip, K.A.3
Potter, M.4
Platt, K.A.5
Oravecz, T.6
-
15
-
-
33144460185
-
Dynamic regulation of pro- and anti-inflammatory cytokines by MAPK phosphatase 1 (MKP-1) in innate immune responses
-
Chi H, Barry SP, Roth RJ, et al: Dynamic regulation of pro- and anti-inflammatory cytokines by MAPK phosphatase 1 (MKP-1) in innate immune responses. Proc Natl Acad Sci USA 103: 2274-2279, 2006.
-
(2006)
Proc Natl Acad Sci USA
, vol.103
, pp. 2274-2279
-
-
Chi, H.1
Barry, S.P.2
Roth, R.J.3
-
16
-
-
0033540056
-
Ghrelin is a growth-hormone-releasing acylated peptide from stomach
-
Kojima M, Hosoda H, Date Y, Nakazato M, Matsuo H and Kangawa K: Ghrelin is a growth-hormone-releasing acylated peptide from stomach. Nature 402: 656-660, 1999.
-
(1999)
Nature
, vol.402
, pp. 656-660
-
-
Kojima, M.1
Hosoda, H.2
Date, Y.3
Nakazato, M.4
Matsuo, H.5
Kangawa, K.6
-
17
-
-
4143111272
-
Upregulation of cardiovascular ghrelin receptor occurs in the hyperdynamic phase of sepsis
-
Wu R, Zhou M, Cui X, Simms HH and Wang P: Upregulation of cardiovascular ghrelin receptor occurs in the hyperdynamic phase of sepsis. Am J Physiol Heart Circ Physiol 287: H1296-H1302, 2004.
-
(2004)
Am J Physiol Heart Circ Physiol
, vol.287
-
-
Wu, R.1
Zhou, M.2
Cui, X.3
Simms, H.H.4
Wang, P.5
-
18
-
-
34247640178
-
Ghrelin down-regulates proinflammatory cytokines in sepsis through activation of the vagus nerve
-
Wu R, Dong W, Cui X, et al: Ghrelin down-regulates proinflammatory cytokines in sepsis through activation of the vagus nerve. Ann Surg 245: 480-486, 2007.
-
(2007)
Ann Surg
, vol.245
, pp. 480-486
-
-
Wu, R.1
Dong, W.2
Cui, X.3
-
19
-
-
26444572289
-
Ghrelin improves tissue perfusion in severe sepsis via down-regulation of endothelin-1
-
Wu R, Dong W, Zhou M, Cui X, Hank Simms H and Wang P: Ghrelin improves tissue perfusion in severe sepsis via down-regulation of endothelin-1. Cardiovasc Res 68: 318-326, 2005.
-
(2005)
Cardiovasc Res
, vol.68
, pp. 318-326
-
-
Wu, R.1
Dong, W.2
Zhou, M.3
Cui, X.4
Hank Simms, H.5
Wang, P.6
-
20
-
-
37149045076
-
Ghrelin inhibits sympathetic nervous activity in sepsis
-
Wu R, Zhou M, Das P, et al: Ghrelin inhibits sympathetic nervous activity in sepsis. Am J Physiol Endocrinol Metab 293: E1697-E1702, 2007.
-
(2007)
Am J Physiol Endocrinol Metab
, vol.293
-
-
Wu, R.1
Zhou, M.2
Das, P.3
-
21
-
-
0031001334
-
Mechanism of hepatocellular dysfunction during early sepsis. Key role of increased gene expression and release of proinflammatory cytokines tumor necrosis factor and interleukin-6
-
Wang P, Ba ZF and Chaudry IH: Mechanism of hepatocellular dysfunction during early sepsis. Key role of increased gene expression and release of proinflammatory cytokines tumor necrosis factor and interleukin-6. Arch Surg 132: 364-370, 1997.
-
(1997)
Arch Surg
, vol.132
, pp. 364-370
-
-
Wang, P.1
Ba, Z.F.2
Chaudry, I.H.3
-
22
-
-
33847176551
-
MAPK phosphatases - regulating the immune response
-
Liu Y, Shepherd EG and Nelin LD: MAPK phosphatases - regulating the immune response. Nat Rev Immunol 7: 202-212, 2007.
-
(2007)
Nat Rev Immunol
, vol.7
, pp. 202-212
-
-
Liu, Y.1
Shepherd, E.G.2
Nelin, L.D.3
-
23
-
-
65549143733
-
MAP kinase phosphatase-1, a critical negative regulator of the innate immune response
-
Li L, Chen SF and Liu Y: MAP kinase phosphatase-1, a critical negative regulator of the innate immune response. Int J Clin Exp Med 2: 48-67, 2009.
-
(2009)
Int J Clin Exp Med
, vol.2
, pp. 48-67
-
-
Li, L.1
Chen, S.F.2
Liu, Y.3
-
24
-
-
0022335639
-
Identification of a set of genes expressed during the G0/G1 transition of cultured mouse cells
-
Lau LF and Nathans D: Identification of a set of genes expressed during the G0/G1 transition of cultured mouse cells. EMBO J 4: 3145-3151, 1985.
-
(1985)
EMBO J
, vol.4
, pp. 3145-3151
-
-
Lau, L.F.1
Nathans, D.2
-
25
-
-
0026779540
-
Oxidative stress and heat shock induce a human gene encoding a protein-tyrosine phosphatase
-
Keyse SM and Emslie EA: Oxidative stress and heat shock induce a human gene encoding a protein-tyrosine phosphatase. Nature 359: 644-647, 1992.
-
(1992)
Nature
, vol.359
, pp. 644-647
-
-
Keyse, S.M.1
Emslie, E.A.2
-
26
-
-
0027254428
-
The growth factor-inducible immediate-early gene 3CH134 encodes a protein-tyrosine-phosphatase
-
Charles CH, Sun H, Lau LF and Tonks NK: The growth factor-inducible immediate-early gene 3CH134 encodes a protein-tyrosine-phosphatase. Proc Natl Acad Sci USA 90: 5292-5296, 1993.
-
(1993)
Proc Natl Acad Sci USA
, vol.90
, pp. 5292-5296
-
-
Charles, C.H.1
Sun, H.2
Lau, L.F.3
Tonks, N.K.4
-
27
-
-
0027306121
-
The human CL100 gene encodes a Tyr/ Thr-protein phosphatase which potently and specifically inactivates MAP kinase and suppresses its activation by oncogenic ras in Xenopus oocyte extracts
-
Alessi DR, Smythe C and Keyse SM: The human CL100 gene encodes a Tyr/ Thr-protein phosphatase which potently and specifically inactivates MAP kinase and suppresses its activation by oncogenic ras in Xenopus oocyte extracts. Oncogene 8: 2015-2020, 1993.
-
(1993)
Oncogene
, vol.8
, pp. 2015-2020
-
-
Alessi, D.R.1
Smythe, C.2
Keyse, S.M.3
-
28
-
-
0027323844
-
Dephosphorylation and inactivation of the mitogen-activated protein kinase by a mitogen-induced Thr/Tyr protein phosphatase
-
Zheng CF and Guan KL: Dephosphorylation and inactivation of the mitogen-activated protein kinase by a mitogen-induced Thr/Tyr protein phosphatase. J Biol Chem 268: 16116-16119, 1993.
-
(1993)
J Biol Chem
, vol.268
, pp. 16116-16119
-
-
Zheng, C.F.1
Guan, K.L.2
-
29
-
-
0027358722
-
MKP-1 (3CH134), an immediate early gene product, is a dual specificity phosphatase that dephosphorylates MAP kinase in vivo
-
Sun H, Charles CH, Lau LF and Tonks NK: MKP-1 (3CH134), an immediate early gene product, is a dual specificity phosphatase that dephosphorylates MAP kinase in vivo. Cell 75: 487-493, 1993.
-
(1993)
Cell
, vol.75
, pp. 487-493
-
-
Sun, H.1
Charles, C.H.2
Lau, L.F.3
Tonks, N.K.4
-
30
-
-
0028924419
-
Role of mitogen-activated protein kinase phosphatase during the cellular response to genotoxic stress. Inhibition of c-Jun N-terminal kinase activity and AP-1-dependent gene activation
-
Liu Y, Gorospe M, Yang C and Holbrook NJ: Role of mitogen-activated protein kinase phosphatase during the cellular response to genotoxic stress. Inhibition of c-Jun N-terminal kinase activity and AP-1-dependent gene activation. J Biol Chem 270: 8377-8380, 1995.
-
(1995)
J Biol Chem
, vol.270
, pp. 8377-8380
-
-
Liu, Y.1
Gorospe, M.2
Yang, C.3
Holbrook, N.J.4
-
31
-
-
0028935270
-
Pro-inflammatory cytokines and environmental stress cause p38 mitogen-activated protein kinase activation by dual phosphorylation on tyrosine and threonine
-
Raingeaud J, Gupta S, Rogers JS, et al: Pro-inflammatory cytokines and environmental stress cause p38 mitogen-activated protein kinase activation by dual phosphorylation on tyrosine and threonine. J Biol Chem 270: 7420-7426, 1995.
-
(1995)
J Biol Chem
, vol.270
, pp. 7420-7426
-
-
Raingeaud, J.1
Gupta, S.2
Rogers, J.S.3
-
32
-
-
0036884621
-
Restraint of proinflammatory cytokine biosynthesis by mitogen-activated protein kinase phosphatase-1 in lipopolysaccharide-stimulated macrophages
-
Chen P, Li J, Barnes J, Kokkonen GC, Lee JC and Liu Y: Restraint of proinflammatory cytokine biosynthesis by mitogen-activated protein kinase phosphatase-1 in lipopolysaccharide-stimulated macrophages. J Immunol 169: 6408-6416, 2002.
-
(2002)
J Immunol
, vol.169
, pp. 6408-6416
-
-
Chen, P.1
Li, J.2
Barnes, J.3
Kokkonen, G.C.4
Lee, J.C.5
Liu, Y.6
-
33
-
-
0038832564
-
Reduced MAP kinase phosphatase-1 degradation after p42/p44MAPK-dependent phosphorylation
-
Brondello JM, Pouyssegur J and McKenzie FR: Reduced MAP kinase phosphatase-1 degradation after p42/p44MAPK-dependent phosphorylation. Science 286: 2514-2517, 1999.
-
(1999)
Science
, vol.286
, pp. 2514-2517
-
-
Brondello, J.M.1
Pouyssegur, J.2
McKenzie, F.R.3
-
34
-
-
0036179521
-
Activation of p42 mitogen-activated protein kinase (MAPK), but not c-Jun NH(2)-terminal kinase, induces phosphorylation and stabilization of MAPK phosphatase XCL100 in Xenopus oocytes
-
Sohaskey ML and Ferrell JE Jr: Activation of p42 mitogen-activated protein kinase (MAPK), but not c-Jun NH(2)-terminal kinase, induces phosphorylation and stabilization of MAPK phosphatase XCL100 in Xenopus oocytes. Mol Biol Cell 13: 454-468, 2002.
-
(2002)
Mol Biol Cell
, vol.13
, pp. 454-468
-
-
Sohaskey, M.L.1
Ferrell Jr, J.E.2
-
35
-
-
45149132474
-
Acetylation of mitogen-activated protein kinase phosphatase-1 inhibits Toll-like receptor signaling
-
Cao W, Bao C, Padalko E and Lowenstein CJ: Acetylation of mitogen-activated protein kinase phosphatase-1 inhibits Toll-like receptor signaling. J Exp Med 205: 1491-1503, 2008.
-
(2008)
J Exp Med
, vol.205
, pp. 1491-1503
-
-
Cao, W.1
Bao, C.2
Padalko, E.3
Lowenstein, C.J.4
|