Endogenous activation of mitochondrial KATP channels protects human failing myocardium from hydroxyl radical-induced stunning

Circulation Research

Volumn 105, Issue 8, 2009, Pages 811-817

  Maack, Christoph ^a,b   Dabew, Ewtim R ^a   Hohl, Mathias ^a   Schäfers, Hans Joachim ^b   Böhm, Michael ^a  

^a Medizinische Klinik und Poliklinik   (Germany)

^b SAARLAND UNIVERSITY   (Germany)

Author keywords

Contractility; Heart failure; Ion channels; Myocardial; Reperfusion; Stunning

Indexed keywords

ADENOSINE TRIPHOSPHATASE (POTASSIUM); GLIBENCLAMIDE; HYDROGEN PEROXIDE; HYDROXIDE; NITRILOTRIACETIC ACID; SULFONYLUREA;

ARTICLE; CELL FUNCTION; CONTROLLED STUDY; DIABETES MELLITUS; DIASTOLIC DYSFUNCTION; ENZYME ACTIVATION; HEART FAILURE; HEART MUSCLE CELL; HEART MUSCLE CONTRACTILITY; HUMAN; HUMAN CELL; HUMAN TISSUE; ISOLATED HEART; PRIORITY JOURNAL; PROTEIN EXPRESSION; SYSTOLIC DYSFUNCTION;

ATRIAL FUNCTION, LEFT; CARDIOMYOPATHY, DILATED; DIABETES COMPLICATIONS; FEMALE; GENE EXPRESSION REGULATION; GLYBURIDE; HEART FAILURE; HEART VENTRICLES; HUMANS; HYDROXYL RADICAL; HYPOGLYCEMIC AGENTS; MALE; MIDDLE AGED; MITOCHONDRIA, HEART; MYOCARDIAL REPERFUSION INJURY; MYOCARDIAL STUNNING; MYOCARDIUM; POTASSIUM CHANNELS, INWARDLY RECTIFYING; SARCOLEMMA;

EID: 70350135479     PISSN: 00097330     EISSN: 15244571     Source Type: Journal    
DOI: 10.1161/CIRCRESAHA.109.206359     Document Type: Article

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