Gallic acid induces neuronal cell death through activation of c-Jun N-terminal kinase and downregulation of Bcl-2

Annals of the New York Academy of Sciences

Volumn 1171, Issue , 2009, Pages 514-520

  Kang, Min Kyung ^a   Kang, Nam Joo ^a,b,c   Jang, Young Jin ^a   Lee, Ki Won ^b   Lee, Hyong Joo ^a  

^a Seoul National University   (South Korea)

^b Konkuk University   (South Korea)

^c Kyungpook National University   (South Korea)

Author keywords

Bcl 2; c Jun N terminal protein kinase; Gallic acid; Resveratrol

Indexed keywords

ANTHRA[1,9 CD]PYRAZOL 6(2H) ONE; ANTIOXIDANT; GALLIC ACID; HYDROGEN PEROXIDE; NICOTINAMIDE ADENINE DINUCLEOTIDE ADENOSINE DIPHOSPHATE RIBOSYLTRANSFERASE; PROTEIN BCL 2; RESVERATROL; STRESS ACTIVATED PROTEIN KINASE;

ANIMAL CELL; ANTIOXIDANT ACTIVITY; APOPTOSIS; CELL PROTECTION; CELL VIABILITY; CONCENTRATION RESPONSE; CONFERENCE PAPER; CONTROLLED STUDY; DOWN REGULATION; DRUG MECHANISM; ENZYME ACTIVATION; ENZYME INHIBITION; ENZYME PHOSPHORYLATION; FRUIT; GENE OVEREXPRESSION; GENETIC TRANSFECTION; GRAPE; NERVE CELL; NERVE CELL NECROSIS; NONHUMAN; OXIDATIVE STRESS; PHEOCHROMOCYTOMA CELL; RAT;

RATTUS; VITACEAE;

EID: 69149098982     PISSN: 00778923     EISSN: 17496632     Source Type: Book Series    
DOI: 10.1111/j.1749-6632.2009.04728.x     Document Type: Conference Paper

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