Overexpression of Hsp20 prevents endotoxin-induced myocardial dysfunction and apoptosis via inhibition of NF-κB activation

Journal of Molecular and Cellular Cardiology

Volumn 47, Issue 3, 2009, Pages 382-390

  Wang, Xiaohong ^a   Zingarelli, Basilia ^b   OConnor, Michael ^b   Zhang, Pengyuan ^a   Adeyemo, Adeola ^a   Kranias, Evangelia G ^a   Wang, Yigang ^a   Fan, Guo Chang ^a  

^a UNIVERSITY OF CINCINNATI COLLEGE OF MEDICINE   (United States)

^b CINCINNATI CHILDREN'S HOSPITAL MEDICAL CENTER   (United States)

Author keywords

Apoptosis; Myocardial function; NF B; Sepsis; Small heat shock protein

Indexed keywords

HEAT SHOCK PROTEIN 20; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; INTERLEUKIN 1BETA; LIPOPOLYSACCHARIDE; TUMOR NECROSIS FACTOR ALPHA;

ANIMAL CELL; ANIMAL EXPERIMENT; APOPTOSIS; ARTICLE; CONTROLLED STUDY; CYTOKINE PRODUCTION; DOWN REGULATION; HEART MUSCLE CELL; HEART MUSCLE CONTRACTILITY; HEART PROTECTION; MALE; MYOCARDIAL DISEASE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; RAT;

ANIMALS; APOPTOSIS; ENDOTOXINS; GENE EXPRESSION REGULATION; HSP20 HEAT-SHOCK PROTEINS; INTERLEUKIN-1BETA; LIPOPOLYSACCHARIDES; MALE; MICE; MICE, TRANSGENIC; MYOCARDIUM; MYOCYTES, CARDIAC; NF-KAPPA B; RATS; RATS, SPRAGUE-DAWLEY; TUMOR NECROSIS FACTOR-ALPHA;

MUS MUSCULUS; RATTUS;

EID: 67650735216     PISSN: 00222828     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.yjmcc.2009.05.016     Document Type: Article

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