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Volumn 30, Issue 1, 2009, Pages 57-64

Deletion of TASK1 and TASK3 channels disrupts intrinsic excitability but does not abolish glucose or pH responses of orexin/hypocretin neurons

Author keywords

Appetite; Firing; Glucose; Hypothalamus; K channels; Mice; Sleep

Indexed keywords

GLUCOSE; GREEN FLUORESCENT PROTEIN; OREXIN; POTASSIUM CHANNEL; POTASSIUM CHANNEL TASK1; POTASSIUM CHANNEL TASK3; UNCLASSIFIED DRUG;

EID: 67649865276     PISSN: 0953816X     EISSN: 14609568     Source Type: Journal    
DOI: 10.1111/j.1460-9568.2009.06789.x     Document Type: Article
Times cited : (55)

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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.