ATP-sensitive potassium channel and mitochondrial permeability transition pore involve in cardioprotection of polydatin

Chinese Journal of Pharmacology and Toxicology

Volumn 23, Issue 2, 2009, Pages 81-88

  Li Ping, Zhang ^a   Chang Ying, Yang ^a   Ying Ping, Wang ^a   Yue, Guan ^a   Ying, X U ^a   Yi, Zhang ^a  

^a HEBEI MEDICAL UNIVERSITY   (China)

Author keywords

Mitochondrial permeability transition pore; Myocardium; Polydatin; Potassium channels; Reper fusion injury

Indexed keywords

5 HYDROXYDECANOIC ACID; ADENOSINE TRIPHOSPHATE SENSITIVE POTASSIUM CHANNEL; ATRACTYLOSIDE; CARDIAC AGENT; GLIBENCLAMIDE; MITOCHONDRIAL PERMEABILITY TRANSITION PORE; POLYDATIN; TRIPHENYLTETRAZOLIUM; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL MEMBRANE PERMEABILITY; CONTROLLED STUDY; DRUG ANTAGONISM; HEART FUNCTION; HEART INFARCTION SIZE; HEART LEFT VENTRICLE ENDDIASTOLIC PRESSURE; HEART MUSCLE ISCHEMIA; HEART PROTECTION; IN VITRO STUDY; MALE; MITOCHONDRIAL PERMEABILITY; NONHUMAN; RAT; REPERFUSION; REPERFUSION INJURY;

RATTUS;

EID: 67049115746     PISSN: 10003002     EISSN: None     Source Type: Journal    
DOI: 10.3867/j.issn.1000-3002.2009.02.001     Document Type: Article

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