Activation of caspase-3 may not contribute to postresuscitation myocardial dysfunction

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 296, Issue 4, 2009, Pages

  Gazmuri, Raul J ^a   Radhakrishnan, Jeejabai ^b   Ayoub, Iyad M ^b  

^a VETERANS AFFAIRS MEDICAL CENTER   (United States)

^b NONE

Author keywords

Apoptosis; Cardiopulmonary resuscitation; Ventricular fibrillation; Ventricular function

Indexed keywords

CARIPORIDE; CASPASE 3; CASPASE 9; CRYSTALLIN; CYTOCHROME C; HEAT SHOCK PROTEIN 70; KETONE DERIVATIVE; SODIUM PROTON EXCHANGE PROTEIN 1; X LINKED INHIBITOR OF APOPTOSIS;

ANIMAL EXPERIMENT; ANIMAL MODEL; APOPTOSIS; ARTICLE; CONTROLLED STUDY; CYTOSOL; DNA FRAGMENTATION; ENZYME ACTIVATION; ENZYME ACTIVITY; ENZYME INHIBITION; HEART PROTECTION; HEART VENTRICLE FIBRILLATION; MALE; MYOCARDIAL DISEASE; NONHUMAN; PRIORITY JOURNAL; PROTEIN CLEAVAGE; RAT; RESUSCITATION;

ANIMALS; ANTI-ARRHYTHMIA AGENTS; APOPTOSIS; CASPASE 3; DISEASE MODELS, ANIMAL; DNA FRAGMENTATION; GUANIDINES; HEART; HEART MASSAGE; MALE; MYOCARDIAL CONTRACTION; MYOCARDIUM; OLIGOPEPTIDES; RATS; RATS, SPRAGUE-DAWLEY; RESUSCITATION; SODIUM-HYDROGEN ANTIPORTER; SULFONES; VENTRICULAR DYSFUNCTION, LEFT; VENTRICULAR FIBRILLATION;

EID: 66249095678     PISSN: 03636135     EISSN: 15221539     Source Type: Journal    
DOI: 10.1152/ajpheart.00338.2008     Document Type: Article

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