Protein product of CLN6 gene responsible for variant late-onset infantile neuronal ceroid lipofuscinosis interacts with CRMP-2

Journal of Neuroscience Research

Volumn 87, Issue 9, 2009, Pages 2157-2166

  Benedict, Jared W ^a   Getty, Amanda L ^a   Wishart, Thomas M ^b   Gillingwater, Thomas H ^b   Pearce, David A ^a  

^a UNIVERSITY OF ROCHESTER SCHOOL OF MEDICINE AND DENTISTRY   (United States)

^b UNIVERSITY OF EDINBURGH   (United Kingdom)

Author keywords

Axon outgrowth; Dihydropyrimidinase like 2 (DRP 2); nclf mouse; Semaphorin 3A

Indexed keywords

COLLAPSIN RESPONSE MEDIATOR PROTEIN 2; MEMBRANE PROTEIN; PROTEIN CLN6; SEMAPHORIN 3A; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; C57BL 6 MOUSE; COCULTURE; CONTROLLED STUDY; FEMALE; GENE MUTATION; GLIA CELL; GROWTH CONE; HIPPOCAMPUS; MALE; MOUSE; NERVE FIBER; NERVE FIBER GROWTH; NEURONAL CEROID LIPOFUSCINOSIS; NONHUMAN; PRIORITY JOURNAL; PROTEIN BLOOD LEVEL; PROTEIN FUNCTION; PROTEIN PROTEIN INTERACTION; SIGNAL TRANSDUCTION; SPINAL GANGLION; THALAMUS;

ANIMALS; BRAIN; CELL DIFFERENTIATION; COCULTURE TECHNIQUES; GANGLIA, SPINAL; GROWTH CONES; HUMANS; INTERCELLULAR SIGNALING PEPTIDES AND PROTEINS; MEMBRANE PROTEINS; MICE; MICE, INBRED C57BL; MICE, MUTANT STRAINS; NERVE DEGENERATION; NERVE TISSUE PROTEINS; NEURAL PATHWAYS; NEUROGENESIS; NEURONAL CEROID-LIPOFUSCINOSES; NIH 3T3 CELLS; SYNAPSES;

EID: 65849386298     PISSN: 03604012     EISSN: 10974547     Source Type: Journal    
DOI: 10.1002/jnr.22032     Document Type: Article

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