APOE 4 polymorphism results in early cognitive deficits in an EAE model

Biochemical and Biophysical Research Communications

Volumn 384, Issue 4, 2009, Pages 466-470

  Tu, Jiang long ^a   Zhao, Chong bo ^a   Vollmer, Timothy ^a   Coons, Stephen ^a   Lin, Hao jie ^a   Marsh, Stephen ^a   Treiman, David M ^a   Shi, Jiong ^a  

^a BARROW NEUROLOGICAL INSTITUTE   (United States)

Author keywords

Apolipoprotein E; Cholinergic; Cognition; EAE; Multiple sclerosis; Water maze

Indexed keywords

APOLIPOPROTEIN E4; CHOLINE ACETYLTRANSFERASE;

ALLERGIC ENCEPHALOMYELITIS; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; COGNITIVE DEFECT; CONTROLLED STUDY; DISEASE MODEL; FEMALE; GENETIC POLYMORPHISM; HIPPOCAMPUS; INFLAMMATION; KNOCKIN MOUSE; KNOCKOUT MOUSE; LEARNING; MOUSE; MULTIPLE SCLEROSIS; NONHUMAN; PRIORITY JOURNAL; PROTEIN PROTEIN INTERACTION; RECALL; SPATIAL ORIENTATION;

ANIMALS; ANIMALS, GENETICALLY MODIFIED; APOLIPOPROTEIN E4; CHOLINE O-ACETYLTRANSFERASE; COGNITION DISORDERS; ENCEPHALOMYELITIS, AUTOIMMUNE, EXPERIMENTAL; GENE KNOCKOUT TECHNIQUES; HIPPOCAMPUS; HUMANS; MEMORY; MULTIPLE SCLEROSIS; POLYMORPHISM, GENETIC; SPATIAL BEHAVIOR;

ANIMALIA; MUS;

EID: 65649103733     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2009.04.153     Document Type: Article

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