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Volumn 182, Issue 6, 2009, Pages 3343-3347

Cutting edge: The PTPN22 allelic variant associated with autoimmunity impairs B cell signaling

Author keywords

[No Author keywords available]

Indexed keywords

BREAKPOINT CLUSTER REGION PROTEIN; NON RECEPTOR PROTEIN TYROSINE PHOSPHATASE 22; LYMPHOCYTE ANTIGEN RECEPTOR; PTPN22 PROTEIN, HUMAN;

EID: 65449176044     PISSN: 00221767     EISSN: 15506606     Source Type: Journal    
DOI: 10.4049/jimmunol.0713370     Document Type: Article
Times cited : (174)

References (16)
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    • Yu, X., J. P. Sun, Y. He, X. Guo, S. Liu, B. Zhou, A. Hudmon, and Z. Y. Zhang. 2007. Structure, inhibitor, and regulatory mechanism of Lyp, a lymphoid-specific tyrosine phosphatase implicated in autoimmune diseases. Proc. Natl. Acad. Sci. USA 104: 19767-19772.
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    • Yu, X.1    Sun, J.P.2    He, Y.3    Guo, X.4    Liu, S.5    Zhou, B.6    Hudmon, A.7    Zhang, Z.Y.8
  • 14
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    • Intrinsic differences in the proliferation of naive and memory human B cells as a mechanism for enhanced secondary immune responses
    • Tangye, S. G., D. T. Avery, E. K. Deenick, and P. D. Hodgkin. 2003. Intrinsic differences in the proliferation of naive and memory human B cells as a mechanism for enhanced secondary immune responses. J. Immunol. 170: 686-694.
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.