Glutathione depletion as a mechanism of 3,4-dideoxyglucosone-3-ene-induced cytotoxicity in human peritoneal mesothelial cells: Role in biocompatibility of peritoneal dialysis fluids

Nephrology Dialysis Transplantation

Volumn 24, Issue 5, 2009, Pages 1436-1442

  Yamamoto, Takashi ^a,b   Tomo, Tadashi ^c   Okabe, Eiji ^c   Namoto, Shinji ^a   Suzuki, Koji ^a   Hirao, Yoshihiko ^b  

^a JMS Co Ltd   (Japan)

^b NARA MEDICAL UNIVERSITY   (Japan)

^c OITA UNIVERSITY   (Japan)

Author keywords

3,4 DGE; CAPD; GDP; Glutathione; Peritoneal mesothelial cell

Indexed keywords

3,4 DIDEOXYGLUCOSON 3 ENE; GLUCOSE DERIVATIVE; GLUTATHIONE; PERITONEAL DIALYSIS FLUID; UNCLASSIFIED DRUG;

ACCURACY; ARTICLE; BIOCOMPATIBILITY; CELL PROLIFERATION; CONTINUOUS AMBULATORY PERITONEAL DIALYSIS; CONTROLLED STUDY; CYTOTOXICITY; ELECTROSPRAY MASS SPECTROMETRY; HIGH PERFORMANCE LIQUID CHROMATOGRAPHY; HUMAN; HUMAN CELL; LIQUID CHROMATOGRAPHY; MESOTHELIUM CELL; OXIDATION REDUCTION STATE; OXIDATIVE STRESS; PERITONEUM; PRIORITY JOURNAL;

APOPTOSIS; CELL PROLIFERATION; CELLS, CULTURED; DIALYSIS SOLUTIONS; DOSE-RESPONSE RELATIONSHIP, DRUG; EPITHELIAL CELLS; GLUTATHIONE; GLYCOGEN PHOSPHORYLASE; HUMANS; OXIDATION-REDUCTION; PERITONEAL DIALYSIS, CONTINUOUS AMBULATORY; PERITONEUM; PYRONES;

EID: 65249172496     PISSN: 09310509     EISSN: 14602385     Source Type: Journal    
DOI: 10.1093/ndt/gfn645     Document Type: Article

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