Genetic instabilities of (CCTG)-(CAGG) and (ATTCT)-(AGAAT) disease-associated repeats reveal multiple pathways for repeat deletion

Molecular Carcinogenesis

Volumn 48, Issue 4, 2009, Pages 336-349

  Edwards, Sharon F ^a   Hashem, Vera I ^b   Klysik, Elzbieta A ^b   Sinden, Richard R ^c  

^a UNIVERSITY OF TEXAS   (United States)

^b TEXAS A AND M HEALTH SCIENCE CENTER   (United States)

^c Florida Institute of Technology   (United States)

Author keywords

Alternative DNA structure; DNA repeat instability; Genomic instability; Repeat deletion; Replication slippage; Sister chromosome exchange

Indexed keywords

MYOTONIC DYSTROPHY PROTEIN KINASE; NUCLEIC ACID BINDING PROTEIN; REPETITIVE DNA; SPINOCEREBELLAR ATAXIA TYPE 10; UNCLASSIFIED DRUG;

ARTICLE; DEGENERATIVE DISEASE; DIMERIZATION; DISEASE ASSOCIATION; DNA REPLICATION; DNA STRAND; DNA STRUCTURE; DNA TEMPLATE; ESCHERICHIA COLI; GENE; GENE DELETION; GENE NUMBER; GENOMIC INSTABILITY; HUMAN; MYOTONIC DYSTROPHY TYPE 1; MYOTONIC DYSTROPHY TYPE 2; PLASMID; PRIORITY JOURNAL; PROTEIN SECONDARY STRUCTURE; RECA GENE; RECB GENE; REGULATORY MECHANISM; SISTER CHROMATID EXCHANGE; GENETICS; GROWTH, DEVELOPMENT AND AGING; METABOLISM; MOLECULAR CLONING; MUTATION; MYOTONIC DYSTROPHY; NUCLEOTIDE REPEAT; SIGNAL TRANSDUCTION; SPINOCEREBELLAR DEGENERATION;

CLONING, MOLECULAR; ESCHERICHIA COLI; GENOMIC INSTABILITY; HUMANS; MUTATION; MYOTONIC DYSTROPHY; PLASMIDS; REPETITIVE SEQUENCES, NUCLEIC ACID; SEQUENCE DELETION; SIGNAL TRANSDUCTION; SPINOCEREBELLAR ATAXIAS;

EID: 65249123648     PISSN: 08991987     EISSN: 10982744     Source Type: Journal    
DOI: 10.1002/mc.20534     Document Type: Article

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