Calcium/calmodulin-dependent protein kinase (CaMK) IV mediates nucleocytoplasmic shuttling and release of HMGB1 during lipopolysaccharide stimulation of macrophages

Journal of Immunology

Volumn 181, Issue 7, 2008, Pages 5015-5023

  Zhang, Xianghong ^a   Wheeler, David ^a   Tang, Ying ^a   Guo, Lanping ^a   Shapiro, Richard A ^a   Ribar, Thomas J ^b   Means, Anthony R ^b   Billiar, Timothy R ^a   Angus, Derek C ^c   Rosengart, Matthew R ^a,c  

^a UNIVERSITY OF PITTSBURGH MEDICAL CENTER   (United States)

^b DUKE UNIVERSITY SCHOOL OF MEDICINE   (United States)

^c UNIVERSITY OF PITTSBURGH SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CALCIUM CALMODULIN DEPENDENT PROTEIN KINASE IV; HIGH MOBILITY GROUP B1 PROTEIN; LIPOPOLYSACCHARIDE; N [2 [[N [3 (4 CHLOROPHENYL) 2 PROPENYL] N METHYLAMINO]METHYL]PHENYL] N (2 HYDROXYETHYL) 4 METHOXYBENZENESULFONAMIDE; PROTEIN SERINE THREONINE KINASE INHIBITOR; SERINE; STO 609; UNCLASSIFIED DRUG; CAMK4 PROTEIN, MOUSE; NUCLEAR PROTEIN;

ANIMAL CELL; ARTICLE; CELL ISOLATION; CELL NUCLEUS; CELL STIMULATION; CELL TYPE; CONTROLLED STUDY; CYTOPLASM; GENE TRANSLOCATION; GROWTH INHIBITION; INCUBATION TIME; MACROPHAGE; NONHUMAN; NUCLEOCYTOPLASMIC TRANSPORT; PERITONEUM MACROPHAGE; PRIORITY JOURNAL; PROTEIN DEPLETION; PROTEIN INTERACTION; PROTEIN PHOSPHORYLATION; PROTEIN SECRETION; RNA INTERFERENCE; SUPERNATANT; ACTIVE TRANSPORT; ANIMAL; CELL CULTURE; CELL LINE; ENZYMOLOGY; IMMUNOLOGY; METABOLISM; MOUSE; PHOSPHORYLATION; PHYSIOLOGY; SECRETION;

ACTIVE TRANSPORT, CELL NUCLEUS; ANIMALS; CALCIUM-CALMODULIN-DEPENDENT PROTEIN KINASE TYPE 4; CELL LINE; CELL NUCLEUS; CELLS, CULTURED; CYTOPLASM; HMGB1 PROTEIN; LIPOPOLYSACCHARIDES; MACROPHAGES, PERITONEAL; MICE; NUCLEAR PROTEINS; PHOSPHORYLATION; SERINE;

EID: 58149308833     PISSN: 00221767     EISSN: 15506606     Source Type: Journal    
DOI: 10.4049/jimmunol.181.7.5015     Document Type: Article

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