Cutting edge: IL-1 controls the IL-23 response induced by gliadin, the etiologic agent in celiac disease

Journal of Immunology

Volumn 181, Issue 7, 2008, Pages 4457-4460

  Harris, Kristina M ^a   Fasano, Alessio ^a   Mann, Dean L ^a,b  

^a UNIVERSITY OF MARYLAND SCHOOL OF MEDICINE   (United States)

^b Med School Teaching Facility 9 00   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

BETA GLUCAN; GLIADIN; HLA DQ2 ANTIGEN; INTERLEUKIN 1; INTERLEUKIN 1BETA; INTERLEUKIN 23; TUMOR NECROSIS FACTOR ALPHA; PEPTIDE FRAGMENT;

ARTICLE; CELIAC DISEASE; CONTROLLED STUDY; CYTOKINE PRODUCTION; CYTOKINE RELEASE; HUMAN; HUMAN CELL; IMMUNOPATHOGENESIS; IMMUNOREGULATION; IMMUNOSTIMULATION; MONOCYTE; PERIPHERAL BLOOD MONONUCLEAR CELL; PRIORITY JOURNAL; SIGNAL TRANSDUCTION; WHEAT; BIOSYNTHESIS; CELL CULTURE; IMMUNOLOGY; METABOLISM; PHYSIOLOGY; PROTEIN SYNTHESIS; SECRETION; UPREGULATION;

CELIAC DISEASE; CELLS, CULTURED; GLIADIN; HUMANS; INTERLEUKIN-1; INTERLEUKIN-1BETA; INTERLEUKIN-23; MONOCYTES; PEPTIDE FRAGMENTS; PROTEIN BIOSYNTHESIS; SIGNAL TRANSDUCTION; UP-REGULATION;

EID: 58149303299     PISSN: 00221767     EISSN: 15506606     Source Type: Journal    
DOI: 10.4049/jimmunol.181.7.4457     Document Type: Article

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