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PACAP produces long-lasting enhancement of synaptic transmission in ganglionic neurons
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Margiotta, J.2
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Depolarization promotes survival of ciliary ganglion neurons by BDNF-dependent and -independent mechanisms
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A null mutation for the alpha3 nicotinic acetylcholine (ACh) receptor gene abolishes fast synaptic activity in sympathetic ganglia and reveals that ACh output from developing preganglionic terminals is regulated in an activity-dependent retrograde manner
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S. Rassadi A. Krishnaswamy B. Pié R. McConnell M. H. Jacob E. Cooper 2005 A null mutation for the alpha3 nicotinic acetylcholine (ACh) receptor gene abolishes fast synaptic activity in sympathetic ganglia and reveals that ACh output from developing preganglionic terminals is regulated in an activity-dependent retrograde manner Journal of Neuroscience 25 8555 8566
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Structural requirements for the occupancy of pituitary adenylate cyclase-activating peptide (PACAP) receptors and adenylate cyclase activation in human neuroblastoma NB-OK-1 cell membranes. Discovery of PACAP(6-38) as a potent antagonist
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P. Robberecht P. Gourlet P. De Neef M.-C. Woussen-Colle M.-C. Vandermeers-Piret A. Vandermeers 1992 Structural requirements for the occupancy of pituitary adenylate cyclase-activating peptide (PACAP) receptors and adenylate cyclase activation in human neuroblastoma NB-OK-1 cell membranes. Discovery of PACAP(6-38) as a potent antagonist European Journal of Biochemistry 207 239 246
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Perspectives on integration of cell extrinsic and cell intrinsic pathways of signaling required for differentiation of noradrenergic sympathetic ganglion neurons
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The diversity of cadherins and implications for a synaptic adhesive code in the CNS
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CREB: A stimulus-induced transcription factor activated by a diverse array of extracellular signals
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A novel cyclic AMP-dependent epac-rit signaling pathway contributes to PACAP38-mediated neuronal differentiation
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Rapid synaptic transmission in the avian ciliary ganglion is mediated by two distinct classes of nicotinic receptors
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