Exaggerated Mg2+ inhibition of Kir2.1 as a consequence of reduced PIP 2 sensitivity in andersen syndrome

Channels

Volumn 1, Issue 3, 2007, Pages 209-217

  Ballester, Leomar Y ^a   Vanoye, Carlos G ^b   George Jr , Alfred L ^a,b  

^a Department of Pharmacology ^*  (United States)

^b VANDERBILT UNIVERSITY   (United States)

Author keywords

Cardiac arrhythmia; Inward rectifier potassium channel; Magnesium; Periodic paralysis; Potassium channel

Indexed keywords

INWARDLY RECTIFYING POTASSIUM CHANNEL; KCNJ2 PROTEIN, HUMAN; MAGNESIUM; PHOSPHATIDYLINOSITOL 4,5 BISPHOSPHATE; PROTEIN SUBUNIT;

ANDERSEN SYNDROME; ANIMAL; ARTICLE; CELL MEMBRANE POTENTIAL; CHO CELL; CRICETULUS; GENETIC TRANSFECTION; GENETICS; HAMSTER; HUMAN; METABOLISM; MISSENSE MUTATION; PROTEIN TERTIARY STRUCTURE; TIME;

ANDERSEN SYNDROME; ANIMALS; CHO CELLS; CRICETINAE; CRICETULUS; HUMANS; MAGNESIUM; MEMBRANE POTENTIALS; MUTATION, MISSENSE; PHOSPHATIDYLINOSITOL 4,5-DIPHOSPHATE; POTASSIUM CHANNELS, INWARDLY RECTIFYING; PROTEIN STRUCTURE, TERTIARY; PROTEIN SUBUNITS; TIME FACTORS; TRANSFECTION;

EID: 53049087281     PISSN: 19336950     EISSN: 19336969     Source Type: Journal    
DOI: 10.4161/chan.4770     Document Type: Article

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