Oxonic acid-induced hyperuricemia elevates plasma aldosterone in experimental renal insufficiency

Journal of Hypertension

Volumn 26, Issue 8, 2008, Pages 1661-1668

  Eräranta, Arttu ^a,b   Kurra, Venla ^c   Tahvanainen, Anna M ^b   Vehmas, Tuija I ^b   Kööbi, Peeter ^b,d   Lakkisto, Päivi ^b,e   Tikkanen, Ilkka ^e,f   Niemelä, Onni J ^d   Mustonen, Jukka T ^b,g   Pörsti, Ilkka H ^b,d,g  

^a Institute of Medical Technology   (Finland)

^b Department of Internal Medicine ^*  (Finland)

^c Medical School   (Finland)

^d TAMPERE UNIVERSITY OF TECHNOLOGY   (Finland)

^e MINERVA FOUNDATION INSTITUTE FOR MEDICAL RESEARCH   (Finland)

^f UNIVERSITY OF HELSINKI   (Finland)

^g TAMPERE UNIVERSITY HOSPITAL   (Finland)

Author keywords

Chronic renal insufficiency; Renin angiotensin aldosterone system; Uric acid

Indexed keywords

ALDOSTERONE; ANGIOTENSIN 1 RECEPTOR; ANGIOTENSIN 2 RECEPTOR; ANGIOTENSIN CONVERTING ENZYME 2; DIPEPTIDYL CARBOXYPEPTIDASE; OXONIC ACID; RENIN; URIC ACID;

ALDOSTERONE BLOOD LEVEL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BLOOD PRESSURE MONITORING; CONTROLLED STUDY; CREATININE CLEARANCE; DISEASE ASSOCIATION; HYPERTENSION; HYPERURICEMIA; KIDNEY FAILURE; MALE; NONHUMAN; PLASMA RENIN ACTIVITY; POTASSIUM EXCRETION; PRIORITY JOURNAL; RAT; RENIN ANGIOTENSIN ALDOSTERONE SYSTEM; SODIUM EXCRETION; URIC ACID BLOOD LEVEL; ANIMAL; ANIMAL FOOD; AUTORADIOGRAPHY; BLOOD; BLOOD PRESSURE; CHEMICALLY INDUCED DISORDER; CHRONIC KIDNEY FAILURE; DISEASE MODEL; GENETICS; METABOLISM; NEPHRECTOMY; PHYSIOLOGY; RENOVASCULAR HYPERTENSION; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; SPRAGUE DAWLEY RAT;

ALDOSTERONE; ANIMAL FEED; ANIMALS; AUTORADIOGRAPHY; BLOOD PRESSURE; DISEASE MODELS, ANIMAL; HYPERTENSION, RENAL; HYPERURICEMIA; MALE; NEPHRECTOMY; OXONIC ACID; RATS; RATS, SPRAGUE-DAWLEY; RECEPTOR, ANGIOTENSIN, TYPE 1; RECEPTOR, ANGIOTENSIN, TYPE 2; RENAL INSUFFICIENCY, CHRONIC; RENIN-ANGIOTENSIN SYSTEM; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION; URIC ACID;

EID: 51549114740     PISSN: 02636352     EISSN: None     Source Type: Journal    
DOI: 10.1097/HJH.0b013e328303205d     Document Type: Article

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