G0 cell cycle arrest alone is insufficient for enabling the repair of ionizing radiation-induced potentially lethal damage

Radiation Research

Volumn 170, Issue 2, 2008, Pages 184-191

  Van Bree, Chris ^a   Rodermond, Hans M ^a   Ten Cate, Rosemarie ^a   De Vos, Judith ^a   Stalpers, Lukas J A ^a   Haveman, Jaap ^a   Medema, Jan Paul ^a   Franken, Nicolaas A P ^a  

^a UNIVERSITY OF AMSTERDAM   (Netherlands)

Author keywords

[No Author keywords available]

Indexed keywords

TRANSCRIPTION FACTOR FKHRL1; CELL CYCLE PROTEIN; FORKHEAD TRANSCRIPTION FACTOR; FOXO3 PROTEIN, MOUSE; GAS1 PROTEIN, MOUSE; MEMBRANE PROTEIN;

ANIMAL CELL; ARTICLE; CARCINOMA CELL; CELL CYCLE ARREST; CELL CYCLE G0 PHASE; CELL PROLIFERATION; CELLULAR DISTRIBUTION; COLON CARCINOMA; CONTROLLED STUDY; FIBROBLAST; IONIZING RADIATION; MOUSE; NONHUMAN; PRIORITY JOURNAL; RADIATION INJURY; RADIATION INJURY REPAIR; TUMOR SUPPRESSOR GENE; ANIMAL; APOPTOSIS; CELL STRAIN 3T3; DNA REPAIR; METABOLISM; PHYSIOLOGY; RADIATION DOSE; RADIATION EXPOSURE; RADIATION RESPONSE;

MURINAE;

ANIMALS; APOPTOSIS; CELL CYCLE PROTEINS; DNA REPAIR; DOSE-RESPONSE RELATIONSHIP, RADIATION; FORKHEAD TRANSCRIPTION FACTORS; G0 PHASE; MEMBRANE PROTEINS; MICE; NIH 3T3 CELLS; RADIATION DOSAGE;

EID: 49249085534     PISSN: 00337587     EISSN: None     Source Type: Journal    
DOI: 10.1667/RR0845.1     Document Type: Article

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