Axonal accumulation of hyperpolarization-activated cyclic nucleotide-gated cation channels contributes to mechanical allodynia after peripheral nerve injury in rat

Pain

Volumn 137, Issue 3, 2008, Pages 495-506

  Jiang, Yu Qiu ^a   Xing, Guo Gang ^a   Wang, Sheng Lan ^a   Tu, Hui Yin ^a   Chi, Ye Nan ^a   Li, Jie ^a   Liu, Feng Yu ^a   Han, Ji Sheng ^a   Wan, You ^a  

^a PEKING UNIVERSITY   (China)

Author keywords

Chronic constriction injury (CCI); Ectopic discharge; Hyperpolarization activated cyclic nucleotide gated cation channel; Ih; Neuropathic pain

Indexed keywords

4 (N ETHYL N PHENYLAMINO) 1,2 DIMETHYL 6 (METHYLAMINO)PYRIMIDINIUM CHLORIDE; CATION CHANNEL; HYPERPOLARIZATION ACTIVATED CYCLIC NUCLEOTIDE GATED CATION CHANNEL;

ALLODYNIA; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; ELECTRON MICROSCOPY; ELECTROPHYSIOLOGY; HYPERALGESIA; IMMUNOHISTOCHEMISTRY; MALE; NERVE FIBER; NEUROPATHIC PAIN; NONHUMAN; PERIPHERAL NERVE INJURY; PRIORITY JOURNAL; PROTEIN EXPRESSION; RAT; SCIATIC NERVE; SPINAL GANGLION; WESTERN BLOTTING;

ANIMALS; AXONS; CYCLIC NUCLEOTIDE-GATED CATION CHANNELS; GANGLIA, SPINAL; HYPERALGESIA; ION CHANNEL GATING; MALE; MEMBRANE POTENTIALS; PERIPHERAL NERVES; POSTERIOR HORN CELLS; RATS; RATS, SPRAGUE-DAWLEY; TOUCH;

EID: 47049086068     PISSN: 03043959     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.pain.2007.10.011     Document Type: Article

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