Selenium diet-supplementation improves cocaine-induced myocardial oxidative stress and prevents cardiac dysfunction in rats

Fundamental and Clinical Pharmacology

Volumn 18, Issue 4, 2004, Pages 431-436

  Moritz, Fabienne ^a   Monteil, Christelle ^a   Isabelle, Marc ^a   Mulder, Paul ^a   Henry, Jean Paul ^a   Derumeaux, Geneviève ^a   Richard, Vincent ^a   Muller, Jean Michel ^b   Thuillez, Christian ^a  

^a INSERM   (France)

^b ROUEN UNIVERSITY HOSPITAL   (France)

Author keywords

Cocaine; Free radicals; Hypertrophy; Selenium; Superoxide; Ventricular function

Indexed keywords

ANTIOXIDANT; COCAINE; FREE RADICAL; GLUTATHIONE PEROXIDASE; SELENIUM; SUPEROXIDE DISMUTASE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CARDIOTOXICITY; COCAINE DEPENDENCE; CONTROLLED STUDY; DIET SUPPLEMENTATION; ECHOCARDIOGRAPHY; ENZYME ACTIVITY; ENZYME ASSAY; HEART FUNCTION TEST; HEART INDEX; HEART LEFT VENTRICLE CONTRACTILITY; HEART LEFT VENTRICLE FUNCTION; HEART LEFT VENTRICLE MASS; MALE; MYOCARDIAL DISEASE; NONHUMAN; OXIDATION REDUCTION STATE; OXIDATIVE STRESS; PRIORITY JOURNAL; RAT; RAT STRAIN;

ANIMALS; COCAINE-RELATED DISORDERS; DIET; GLUTATHIONE PEROXIDASE; MALE; OXIDATION-REDUCTION; OXIDATIVE STRESS; RATS; RATS, WISTAR; SELENIUM; SUPEROXIDE DISMUTASE; VENTRICULAR DYSFUNCTION, LEFT;

EID: 4544388205     PISSN: 07673981     EISSN: None     Source Type: Journal    
DOI: 10.1111/j.1472-8206.2004.00255.x     Document Type: Article

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