Additional lack of iNOS attenuates diastolic dysfunction in aged ET-1 transgenic mice

Canadian Journal of Physiology and Pharmacology

Volumn 86, Issue 6, 2008, Pages 353-357

  Kalk, Philipp ^a,b   Westermann, Dirk ^b   Herzfeld, Sophia ^a   Relle, Katharina ^a,b   Pfab, Thiemo ^b   Bauer, Christian ^c   Tschöpe, Carsten ^b   Stasch, Johannes Peter ^d,e   Hocher, Berthold ^a  

^a DZHK GERMAN CENTRE FOR CARDIOVASCULAR RESEARCH   (Germany)

^b CHARITÉ UNIVERSITÄTSMEDIZIN BERLIN   (Germany)

^c FREIE UNIVERSITÄT BERLIN   (Germany)

^d Cardiovascular Research   (Germany)

^e MARTIN LUTHER UNIVERSITY HALLE WITTENBERG   (Germany)

Author keywords

Cardiac injury; Endothelin; NO

Indexed keywords

BRAIN NATRIURETIC PEPTIDE; ENDOTHELIN 1; INDUCIBLE NITRIC OXIDE SYNTHASE;

AGING; ANIMAL TISSUE; CONFERENCE PAPER; CONTROLLED STUDY; CROSS BREEDING; DIASTOLIC DYSFUNCTION; ENZYME ACTIVATION; ENZYME DEFICIENCY; GENOTYPE; HEART CATHETERIZATION; HEART HEMODYNAMICS; HEART INJURY; HEART LEFT VENTRICLE FUNCTION; HEART MUSCLE CELL; HEART MUSCLE FIBROSIS; HEART OUTPUT; HEART WEIGHT; HISTOLOGY; HISTOPATHOLOGY; KNOCKOUT MOUSE; MOUSE; MUSCLE CELL; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; SYSTOLIC HEART FAILURE; TRANSGENIC MOUSE; WILD TYPE;

AGING; ANIMALS; DIASTOLE; ENDOTHELIN-1; MICE; MICE, KNOCKOUT; MICE, TRANSGENIC; MYOCARDIUM; NATRIURETIC PEPTIDE, BRAIN; NITRIC OXIDE SYNTHASE TYPE II; VENTRICULAR DYSFUNCTION, LEFT;

EID: 44949145343     PISSN: 00084212     EISSN: None     Source Type: Journal    
DOI: 10.1139/Y08-032     Document Type: Conference Paper

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