Loss of collagen XVIII enhances neovascularization and vascular permeability in atherosclerosis

Circulation

Volumn 110, Issue 10, 2004, Pages 1330-1336

  Moulton, Karen S ^a,b   Olsen, Bjorn R ^c   Sonn, Silvia ^a   Fukai, Naomi ^c   Zurakowski, David ^a   Zeng, Xiaokun ^a  

^a BOSTON CHILDREN S HOSPITAL   (United States)

^b BRIGHAM AND WOMEN S HOSPITAL   (United States)

^c HARVARD MEDICAL SCHOOL   (United States)

Author keywords

Angiogenesis; Atherosclerosis; Extracellular matrix; Permeability; Vasa vasorum

Indexed keywords

APOLIPOPROTEIN E; COLLAGEN TYPE 18; ENDOSTATIN; PROTEOGLYCAN;

ANGIOGENESIS; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; ATHEROSCLEROSIS; BASEMENT MEMBRANE; BLOOD VESSEL PERMEABILITY; CONCENTRATION RESPONSE; CONTROLLED STUDY; CORRELATION ANALYSIS; ENDOTHELIUM CELL; HETEROZYGOSITY; KNOCKOUT MOUSE; MOUSE; NEOVASCULARIZATION (PATHOLOGY); NONHUMAN; PATHOPHYSIOLOGY; PRIORITY JOURNAL; PROTEIN FUNCTION; RAT;

ANIMALS; AORTIC DISEASES; APOLIPOPROTEINS E; ATHEROSCLEROSIS; CAPILLARY PERMEABILITY; CHOLESTEROL, DIETARY; COLLAGEN TYPE XVIII; DIET, ATHEROGENIC; ENDOTHELIUM, VASCULAR; GENE DOSAGE; LIPID METABOLISM; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; NEOVASCULARIZATION, PATHOLOGIC; ORGAN CULTURE TECHNIQUES; SKIN; VASA VASORUM; VASCULAR ENDOTHELIAL GROWTH FACTOR A;

EID: 4444257284     PISSN: 00097322     EISSN: None     Source Type: Journal    
DOI: 10.1161/01.CIR.0000140720.79015.3C     Document Type: Article

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