Molecular mechanisms of Cr(VI)-induced carcinogenesis

Molecular and Cellular Biochemistry

Volumn 234-235, Issue , 2002, Pages 293-300

  Ding, Min ^a   Shi, Xianglin ^a  

^a NATIONAL INSTITUTE FOR OCCUPATIONAL SAFETY AND HEALTH   (United States)

Author keywords

Apoptosis; Carcinogenesis; Cell growth arrest; Cr(VI); Reactive oxygen species; Signal transduction; Transcriptional regulation

Indexed keywords

ASCORBIC ACID; CHROMIC ACID; FLAVOPROTEIN; FREE RADICAL; HYPOXIA INDUCIBLE FACTOR 1; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; PROTEIN P53; THIOL DERIVATIVE; TRANSCRIPTION FACTOR AP 1; VASCULOTROPIN; CARCINOGEN; CHROMIUM; CHROMIUM HEXAVALENT ION;

APOPTOSIS; ARTICLE; CARCINOGENESIS; CELL GROWTH; CELL ORGANELLE; CHEMICAL REACTION; CONTROLLED STUDY; DNA DAMAGE; GENE EXPRESSION; GROWTH INHIBITION; HUMAN; NONHUMAN; OXIDATION; PROTEIN PHOSPHORYLATION; REDUCTION; ANIMAL; CELL CYCLE; CELL TRANSFORMATION; DRUG EFFECT; GENE EXPRESSION REGULATION; METABOLISM; OXIDATION REDUCTION REACTION; REVIEW;

ANIMAL; CARCINOGENS; CELL CYCLE; CELL TRANSFORMATION, NEOPLASTIC; CHROMIUM; DNA DAMAGE; FREE RADICALS; GENE EXPRESSION REGULATION, NEOPLASTIC; HUMAN; OXIDATION-REDUCTION; ANIMALS; HUMANS;

EID: 4243735675     PISSN: 03008177     EISSN: None     Source Type: Journal    
DOI: 10.1023/A:1015975218920     Document Type: Article

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