Endothelial nitric oxide synthase decreases β-adrenergic responsiveness via inhibition of the L-type Ca2+ current

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 294, Issue 3, 2008, Pages

  Wang, Honglan ^a   Kohr, Mark J ^a   Wheeler, Debra G ^a   Ziolo, Mark T ^a  

^a OHIO STATE UNIVERSITY   (United States)

Author keywords

Action potential; Cardiac myocytes; Early afterdepolarizations; Endothelial nitric oxide synthase

Indexed keywords

CALCIUM CHANNEL L TYPE; ENDOTHELIAL NITRIC OXIDE SYNTHASE; ISOPRENALINE;

ACTION POTENTIAL; ADRENERGIC SYSTEM; AMPLITUDE MODULATION; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; ENZYME INHIBITION; HEART ARRHYTHMIA; HEART DEPOLARIZATION; HEART FAILURE; HEART MUSCLE CELL; HEART PROTECTION; INHIBITION KINETICS; KNOCKOUT MOUSE; MEASUREMENT; MOUSE; NONHUMAN; PATCH CLAMP; PRIORITY JOURNAL; SIGNAL TRANSDUCTION; WILD TYPE;

ACTION POTENTIALS; ADRENERGIC BETA-AGONISTS; ANIMALS; ARRHYTHMIAS, CARDIAC; CALCIUM CHANNELS, L-TYPE; CALCIUM SIGNALING; ISOPROTERENOL; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MYOCARDIAL CONTRACTION; MYOCYTES, CARDIAC; NITRIC OXIDE SYNTHASE TYPE III; PATCH-CLAMP TECHNIQUES; RECEPTORS, ADRENERGIC, BETA;

EID: 41549122317     PISSN: 03636135     EISSN: 15221539     Source Type: Journal    
DOI: 10.1152/ajpheart.01249.2007     Document Type: Article

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