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Volumn 32, Issue 3, 2008, Pages 508-510

A recurrent splicing variant without c-ABL Exon 7 in Imatinib-resistant patients

Author keywords

[No Author keywords available]

Indexed keywords

ABELSON KINASE; AMINO ACID; BCR ABL PROTEIN; COMPLEMENTARY DNA; IMATINIB; NUCLEOTIDE; RNA; TUMOR MARKER;

EID: 39749147983     PISSN: 01452126     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.leukres.2007.04.018     Document Type: Letter
Times cited : (12)

References (10)
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  • 2
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  • 3
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    • Clinical resistance to STI-571 cancer therapy caused by BCR-ABL gene mutation or amplification
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  • 4
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    • Preexistence and evolution of imatinib mesylate-resistant clones in chronic myelogenous leukemia detected by a PNA-based PCR clamping technique
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  • 5
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    • Monitoring patients in complete cytogenetic remission after treatment of CML in chronic phase with imatinib: patterns of residual leukaemia and prognostic factors for cytogenetic relapse
    • Marin D., Kaeda J., Szydlo R., Saunders S., Fleming A., Howard J., et al. Monitoring patients in complete cytogenetic remission after treatment of CML in chronic phase with imatinib: patterns of residual leukaemia and prognostic factors for cytogenetic relapse. Leukemia 19 (2005) 507-512
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  • 6
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    • A novel BCR-ABL splice isoform is associated with the L248V mutation in CML patients with acquired resistance to imatinib
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.