The influence of insulin growth factor-I on the apoptosis of cardiomyocytes subjected to ischemia and hypoxia

Chinese Journal of Burns

Volumn 23, Issue 6, 2007, Pages 436-439

  Song, Hua Pei ^a   Yan, Hong ^a   Zhang, Dong Xia ^a   Chu, Zhi Gang ^a   Zhang, Qiong ^a   Huang, Yue Sheng ^a  

^a SOUTHWEST HOSPITAL   (China)

Author keywords

Cell hypoxia; Insulin like growth factor 1 Apoptosis; Membrane potentials; Myocyte, cardiac

Indexed keywords

PHOSPHATIDYLINOSITOL 3 KINASE; PROTEIN KINASE B; SOMATOMEDIN C;

ANIMAL CELL; APOPTOSIS; ARTICLE; CONTROLLED STUDY; ENZYME ACTIVATION; ENZYME LINKED IMMUNOSORBENT ASSAY; HEART CELL CULTURE; HEART MITOCHONDRION; HEART MUSCLE CELL; HEART MUSCLE FIBER MEMBRANE POTENTIAL; HEART MUSCLE ISCHEMIA; IN VITRO STUDY; LASER MICROSCOPY; MITOCHONDRIAL MEMBRANE; NONHUMAN; PATHOGENESIS; RAT; SIGNAL TRANSDUCTION; STATISTICAL SIGNIFICANCE; WESTERN BLOTTING;

ANIMALS; ANOXIA; APOPTOSIS; CELL HYPOXIA; CELLS, CULTURED; INSULIN-LIKE GROWTH FACTOR I; ISCHEMIA; MEMBRANE POTENTIAL, MITOCHONDRIAL; MYOCYTES, CARDIAC; PROTO-ONCOGENE PROTEINS C-AKT; RANDOM ALLOCATION; RATS; RATS, SPRAGUE-DAWLEY;

EID: 39449113104     PISSN: 10092587     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (12)

1. Goldspink G. Mechanical signals, IGF- I gene splicing, and muscle adaptation. Physiology, 2005, 20(10): 232-238.
2. Yang SY, Goldspink G. Different roles of the IGF- I Ec peptide (MGF) and mature IGF- I in myoblast proliferation and differentiation. FEBS,2002,522 (1/2/3):156-160.
3. Chinese source.
4. Chinese source.
5. Wajant H. The fas signaling pathway: more than a paradigm. Science,2002,296(31):1635-1636.
6. Lugli E, Troiano L, Ferraresi R, et al. Characterization of cells with different mitochondrial membrane potential during apoptosis. Cytometry A,2005,68(2):28-35.
7. Düßmann H, Rehm M, Donat K, et al. Outer mitochondrial membrane permeabilization during apoptosis triggers caspase-independent mitochondrial and caspase-dependent plasma membrane potential depolarization: a single-cell analysis. J Cell Sci,2002, 116(11):525-536.
8. Kazimierz W, Baran J, Zembala M, et al. Caspase-8 activation precedes alterations of mitochondrial membrane potential during monocyte apoptosis induced by phagocytosis and killing of staphylococcus aureus. Infect Immun, 2004, 72 (5): 2590-2597.
9. Russell C, Scaduto J, Lee W, et al. Measurement of mitochondrial membrane potential using fluorescent rhodaminederivatives. Biophys J,1999, 76(4):469-477.
10. Gottlieb E, Armour SM, Thompson CB. Mitochondrial membrane potential regulates matrix configuration and cytochrome c release during apoptosis. Cell Death Differ,2003,10(6):709-717.
11. Hirai K, Hayashi T, Chan PH. PI3K inhibition in neonatal rat brain slices during and after hypoxia reduces phospho-Akt and increases cytosolic cytochrome c and apoptosis. Brain Res Mol Brain Res, 2004,124(1):51-61.
12. Bergmann A. Survival signaling goes BAD. Dev Cell, 2002,3 (1): 607-608.