JNK/p38 MAPK involves in ginsenoside Rb1 attenuating beta-amyloid peptide (25-35)-induced tau protein hyperphosphorylation in embryo rat cortical neurons

Yaoxue Xuebao

Volumn 43, Issue 1, 2008, Pages 29-34

  Song, Jin Qiu ^a   Chen, Xiao Chun ^a   Zhang, Jing ^a   Huang, Tian Wen ^a   Zeng, Yu Qi ^a   Shen, Jie ^a   Chen, Li Min ^a  

^a FUJIAN MEDICAL UNIVERSITY   (China)

Author keywords

Beta amyloid peptide (25 35); Ginsenoside Rb1; JNK p38 MAPK; Phosphorylation; Tau protein

Indexed keywords

AMYLOID BETA PROTEIN; AMYLOID BETA PROTEIN (25 35); AMYLOID BETA-PROTEIN (25-35); GINSENOSIDE; GINSENOSIDE RB 1; MITOGEN ACTIVATED PROTEIN KINASE P38; PEPTIDE FRAGMENT; STRESS ACTIVATED PROTEIN KINASE; TAU PROTEIN;

ANIMAL; ARTICLE; BRAIN CORTEX; CELL CULTURE; CHEMISTRY; CYTOLOGY; DRUG ANTAGONISM; GINSENG; ISOLATION AND PURIFICATION; MEDICINAL PLANT; METABOLISM; NERVE CELL; PHOSPHORYLATION; RAT; SIGNAL TRANSDUCTION; SPRAGUE DAWLEY RAT;

AMYLOID BETA-PROTEIN; ANIMALS; CELLS, CULTURED; CEREBRAL CORTEX; GINSENOSIDES; JNK MITOGEN-ACTIVATED PROTEIN KINASES; NEURONS; P38 MITOGEN-ACTIVATED PROTEIN KINASES; PANAX; PEPTIDE FRAGMENTS; PHOSPHORYLATION; PLANTS, MEDICINAL; RATS; RATS, SPRAGUE-DAWLEY; SIGNAL TRANSDUCTION; TAU PROTEINS;

EID: 38849088159     PISSN: 05134870     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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