-
1
-
-
15844401780
-
Expression of caveolin-3 in skeletal, cardiac, and smooth muscle cells. Caveolin-3 is a component of the sarcolemma and co-fractionates with dystrophin and dystrophin-associated glycoproteins
-
Song K.S., Scherer P.E., Tang Z., Okamoto T., Li S., Chafel M., Chu C., Kohtz D.S., and Lisanti M.P. Expression of caveolin-3 in skeletal, cardiac, and smooth muscle cells. Caveolin-3 is a component of the sarcolemma and co-fractionates with dystrophin and dystrophin-associated glycoproteins. J. Biol. Chem. 271 (1996) 15160-15165
-
(1996)
J. Biol. Chem.
, vol.271
, pp. 15160-15165
-
-
Song, K.S.1
Scherer, P.E.2
Tang, Z.3
Okamoto, T.4
Li, S.5
Chafel, M.6
Chu, C.7
Kohtz, D.S.8
Lisanti, M.P.9
-
2
-
-
2142647335
-
The biology of caveolae: lessons from caveolin knockout mice and implications for human disease
-
(Review)
-
Hnasko R., and Lisanti M.P. The biology of caveolae: lessons from caveolin knockout mice and implications for human disease. Mol. Interv. 3 (2003) 8445-8464 (Review)
-
(2003)
Mol. Interv.
, vol.3
, pp. 8445-8464
-
-
Hnasko, R.1
Lisanti, M.P.2
-
3
-
-
1342267006
-
Caveolinopathies: mutations in caveolin-3 cause four distinct autosomal dominant muscle diseases
-
(Review)
-
Woodman S.E., Sotgia F., Galbiati F., Minetti C., and Lisanti M.P. Caveolinopathies: mutations in caveolin-3 cause four distinct autosomal dominant muscle diseases. Neurology 62 (2004) 538-543 (Review)
-
(2004)
Neurology
, vol.62
, pp. 538-543
-
-
Woodman, S.E.1
Sotgia, F.2
Galbiati, F.3
Minetti, C.4
Lisanti, M.P.5
-
4
-
-
0033569755
-
Targeted down-regulation of caveolin-3 is sufficient to inhibit myotube formation in differentiating C2C12 myoblasts. Transient activation of p38 mitogen-activated protein kinase is required for induction of caveolin-3 expression and subsequent myotube formation
-
Galbiati F., Volonte D., Engelman J.A., Scherer P.E., and Lisanti M.P. Targeted down-regulation of caveolin-3 is sufficient to inhibit myotube formation in differentiating C2C12 myoblasts. Transient activation of p38 mitogen-activated protein kinase is required for induction of caveolin-3 expression and subsequent myotube formation. J. Biol. Chem. 274 (1999) 30315-30321
-
(1999)
J. Biol. Chem.
, vol.274
, pp. 30315-30321
-
-
Galbiati, F.1
Volonte, D.2
Engelman, J.A.3
Scherer, P.E.4
Lisanti, M.P.5
-
5
-
-
34147128560
-
Hypertrophy and atrophy inversely regulate Caveolin-3 expression in myoblasts
-
Fanzani A., Musaro A., Stoppani E., Giuliani R., Colombo F., Preti A., and Marchesini S. Hypertrophy and atrophy inversely regulate Caveolin-3 expression in myoblasts. Biochem. Biophys. Res. Commun. 357 (2007) 314-318
-
(2007)
Biochem. Biophys. Res. Commun.
, vol.357
, pp. 314-318
-
-
Fanzani, A.1
Musaro, A.2
Stoppani, E.3
Giuliani, R.4
Colombo, F.5
Preti, A.6
Marchesini, S.7
-
6
-
-
0031920515
-
Mutations in the caveolin-3 gene cause autosomal dominant limb-girdle muscular dystrophy
-
Minetti C., Sotgia F., Bruno C., Scartezzini P., Broda P., Bado M., Masetti E., Mazzocco P., Egeo A., Donati M.A., Volonté D., Galbiati F., Cordone G., Bricarelli F.D., Lisanti M.P., and Zara F. Mutations in the caveolin-3 gene cause autosomal dominant limb-girdle muscular dystrophy. Nat. Genet. 18 (1998) 365-368
-
(1998)
Nat. Genet.
, vol.18
, pp. 365-368
-
-
Minetti, C.1
Sotgia, F.2
Bruno, C.3
Scartezzini, P.4
Broda, P.5
Bado, M.6
Masetti, E.7
Mazzocco, P.8
Egeo, A.9
Donati, M.A.10
Volonté, D.11
Galbiati, F.12
Cordone, G.13
Bricarelli, F.D.14
Lisanti, M.P.15
Zara, F.16
-
7
-
-
0034531315
-
Limb-girdle muscular dystrophy (LGMD-1C) mutants of caveolin-3 undergo ubiquitination and proteasomal degradation
-
Galbiati F., Volonte D., Minetti C., Bregman D.B., and Lisanti M.P. Limb-girdle muscular dystrophy (LGMD-1C) mutants of caveolin-3 undergo ubiquitination and proteasomal degradation. J. Biol. Chem. 275 (2000) 37702-37711
-
(2000)
J. Biol. Chem.
, vol.275
, pp. 37702-37711
-
-
Galbiati, F.1
Volonte, D.2
Minetti, C.3
Bregman, D.B.4
Lisanti, M.P.5
-
8
-
-
0035253580
-
Transgenic mice expressing mutant caveolin-3 show severe myopathy associated with increased nNOS activity
-
Sunada Y., Ohi H., Hase A., Ohi H., Hosono T., Arata S., Higuchi S., Matsumura K., and Shimizu T. Transgenic mice expressing mutant caveolin-3 show severe myopathy associated with increased nNOS activity. Hum. Mol. Genet. 10 (2001) 173-178
-
(2001)
Hum. Mol. Genet.
, vol.10
, pp. 173-178
-
-
Sunada, Y.1
Ohi, H.2
Hase, A.3
Ohi, H.4
Hosono, T.5
Arata, S.6
Higuchi, S.7
Matsumura, K.8
Shimizu, T.9
-
9
-
-
33750578365
-
Muscular atrophy of caveolin-3-deficient mice is rescued by myostatin inhibition
-
Ohsawa Y., Hagiwara H., Nakatani M., Yasue A., Moriyama K., Murakami T., Tsuchida K., Noji S., and Sunada Y. Muscular atrophy of caveolin-3-deficient mice is rescued by myostatin inhibition. J. Clin. Invest. 116 (2006) 2924-2934
-
(2006)
J. Clin. Invest.
, vol.116
, pp. 2924-2934
-
-
Ohsawa, Y.1
Hagiwara, H.2
Nakatani, M.3
Yasue, A.4
Moriyama, K.5
Murakami, T.6
Tsuchida, K.7
Noji, S.8
Sunada, Y.9
-
10
-
-
24144480448
-
Zebrafish as a model for caveolin-associated muscle disease; caveolin-3 is required for myofibril organization and muscle cell patterning
-
Nixon S.J., Wegner J., Ferguson C., Mery P.F., Hancock J.F., Currie P.D., Key B., Westerfield M., and Parton R.G. Zebrafish as a model for caveolin-associated muscle disease; caveolin-3 is required for myofibril organization and muscle cell patterning. Hum. Mol. Genet. 14 (2005) 1727-1743
-
(2005)
Hum. Mol. Genet.
, vol.14
, pp. 1727-1743
-
-
Nixon, S.J.1
Wegner, J.2
Ferguson, C.3
Mery, P.F.4
Hancock, J.F.5
Currie, P.D.6
Key, B.7
Westerfield, M.8
Parton, R.G.9
-
11
-
-
0346121432
-
A caveolin-3 mutant that causes limb girdle muscular dystrophy type 1C disrupts Src localization and activity and induces apoptosis in skeletal myotubes
-
Smythe G.M., Eby J.C., Disatnik M.H., and Rando T.A. A caveolin-3 mutant that causes limb girdle muscular dystrophy type 1C disrupts Src localization and activity and induces apoptosis in skeletal myotubes. J. Cell Sci. 116 (2003) 4739-4749
-
(2003)
J. Cell Sci.
, vol.116
, pp. 4739-4749
-
-
Smythe, G.M.1
Eby, J.C.2
Disatnik, M.H.3
Rando, T.A.4
-
12
-
-
33747362294
-
Altered caveolin-3 expression disrupts PI(3) kinase signaling leading to death of cultured muscle cells
-
Smythe G.M., and Rando T.A. Altered caveolin-3 expression disrupts PI(3) kinase signaling leading to death of cultured muscle cells. Exp. Cell Res. 312 (2006) 2816-2825
-
(2006)
Exp. Cell Res.
, vol.312
, pp. 2816-2825
-
-
Smythe, G.M.1
Rando, T.A.2
-
13
-
-
0035736260
-
Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo
-
Bodine S.C., Stitt T.N., Gonzalez M., Kline W.O., Stover G.L., Bauerlein R., Zlotchenko E., Scrimgeour A., Lawrence J.C., Glass D.J., and Yancopoulos G.D. Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo. Nat. Cell Biol. 3 (2001) 1014-1019
-
(2001)
Nat. Cell Biol.
, vol.3
, pp. 1014-1019
-
-
Bodine, S.C.1
Stitt, T.N.2
Gonzalez, M.3
Kline, W.O.4
Stover, G.L.5
Bauerlein, R.6
Zlotchenko, E.7
Scrimgeour, A.8
Lawrence, J.C.9
Glass, D.J.10
Yancopoulos, G.D.11
-
14
-
-
0038624395
-
Dwarfism, impaired skin development, skeletal muscle atrophy, delayed bone development, and impeded adipogenesis in mice lacking Akt1 and Akt2
-
Peng X.D., Xu P.Z., Chen M.L., Hahn-Windgassen A., Skeen J., Jacobs J., Sundararajan D., Chen W.S., Crawford S.E., Coleman K.G., and Hay N. Dwarfism, impaired skin development, skeletal muscle atrophy, delayed bone development, and impeded adipogenesis in mice lacking Akt1 and Akt2. Genes Dev. 17 (2003) 1352-1365
-
(2003)
Genes Dev.
, vol.17
, pp. 1352-1365
-
-
Peng, X.D.1
Xu, P.Z.2
Chen, M.L.3
Hahn-Windgassen, A.4
Skeen, J.5
Jacobs, J.6
Sundararajan, D.7
Chen, W.S.8
Crawford, S.E.9
Coleman, K.G.10
Hay, N.11
-
15
-
-
6344256238
-
Conditional activation of akt in adult skeletal muscle induces rapid hypertrophy
-
Lai K.M., Gonzalez M., Poueymirou W.T., Kline W.O., Na E., Zlotchenko E., Stitt T.N., Economides A.N., Yancopoulos G.D., and Glass D.J. Conditional activation of akt in adult skeletal muscle induces rapid hypertrophy. Mol. Cell Biol. 24 (2004) 9295-9304
-
(2004)
Mol. Cell Biol.
, vol.24
, pp. 9295-9304
-
-
Lai, K.M.1
Gonzalez, M.2
Poueymirou, W.T.3
Kline, W.O.4
Na, E.5
Zlotchenko, E.6
Stitt, T.N.7
Economides, A.N.8
Yancopoulos, G.D.9
Glass, D.J.10
-
16
-
-
2342627231
-
Deacetylase inhibitors increase muscle cell size by promoting myoblast recruitment and fusion through induction of follistatin
-
Iezzi S., Di Padova M., Serra C., Caretti G., Simone C., Maklan E., Minetti G., Zhao P., Hoffman E.P., Puri P.L., and Sartorelli V. Deacetylase inhibitors increase muscle cell size by promoting myoblast recruitment and fusion through induction of follistatin. Dev Cell. 6 (2004) 673-684
-
(2004)
Dev Cell.
, vol.6
, pp. 673-684
-
-
Iezzi, S.1
Di Padova, M.2
Serra, C.3
Caretti, G.4
Simone, C.5
Maklan, E.6
Minetti, G.7
Zhao, P.8
Hoffman, E.P.9
Puri, P.L.10
Sartorelli, V.11
-
17
-
-
0037726553
-
IL-4 acts as a myoblast recruitment factor during mammalian muscle growth
-
Horsley V., Jansen K.M., Mills S.T., and Pavlath G.K. IL-4 acts as a myoblast recruitment factor during mammalian muscle growth. Cell 113 (2003) 483-494
-
(2003)
Cell
, vol.113
, pp. 483-494
-
-
Horsley, V.1
Jansen, K.M.2
Mills, S.T.3
Pavlath, G.K.4
-
18
-
-
33745856252
-
Hypertrophic response of Duchenne and limb-girdle muscular dystrophies is associated with activation of Akt pathway
-
Peter A.K., and Crosbie R.H. Hypertrophic response of Duchenne and limb-girdle muscular dystrophies is associated with activation of Akt pathway. Exp. Cell Res. 312 (2006) 2580-2591
-
(2006)
Exp. Cell Res.
, vol.312
, pp. 2580-2591
-
-
Peter, A.K.1
Crosbie, R.H.2
-
19
-
-
0035941020
-
Identification of ubiquitin ligases required for skeletal muscle atrophy
-
Bodine S.C., Latres E., Baumhueter S., Lai V.K., Nunez L., Clarke B.A., Poueymirou F.J., Panaro E.Na.K., Dharmarajan Z.Q., Pan D.M., Valenzuela T.M., DeChiara W.T., Stitt T.N., Yancopoulos G.D., and Glass D.J. Identification of ubiquitin ligases required for skeletal muscle atrophy. Science 294 (2001) 1704-1708
-
(2001)
Science
, vol.294
, pp. 1704-1708
-
-
Bodine, S.C.1
Latres, E.2
Baumhueter, S.3
Lai, V.K.4
Nunez, L.5
Clarke, B.A.6
Poueymirou, F.J.7
Panaro, E.Na.K.8
Dharmarajan, Z.Q.9
Pan, D.M.10
Valenzuela, T.M.11
DeChiara, W.T.12
Stitt, T.N.13
Yancopoulos, G.D.14
Glass, D.J.15
-
20
-
-
0035807969
-
Atrogin-1, a muscle-specific F-box protein highly expressed during muscle atrophy
-
Gomes M.D., Lecker S.H., Jagoe R.T., Navon A., and Goldberg A.L. Atrogin-1, a muscle-specific F-box protein highly expressed during muscle atrophy. Proc. Natl. Acad. Sci. USA 98 (2001) 14440-14445
-
(2001)
Proc. Natl. Acad. Sci. USA
, vol.98
, pp. 14440-14445
-
-
Gomes, M.D.1
Lecker, S.H.2
Jagoe, R.T.3
Navon, A.4
Goldberg, A.L.5
-
21
-
-
0347285363
-
Multiple types of skeletal muscle atrophy involve a common program of changes in gene expression
-
Lecker S.H., Jagoe R.T., Gilbert A., Gomes M., Baracos V., Bailey J., Price S.R., Mitch W.E., and Goldberg A.L. Multiple types of skeletal muscle atrophy involve a common program of changes in gene expression. FASEB J. 18 (2004) 39-51
-
(2004)
FASEB J.
, vol.18
, pp. 39-51
-
-
Lecker, S.H.1
Jagoe, R.T.2
Gilbert, A.3
Gomes, M.4
Baracos, V.5
Bailey, J.6
Price, S.R.7
Mitch, W.E.8
Goldberg, A.L.9
-
22
-
-
11144356337
-
Foxo transcription factors induce the atrophy-related ubiquitin ligase atrogin-1 and cause skeletal muscle atrophy
-
Sandri M., Sandri C., Gilbert A., Skurk C., Calabria E., Picard A., Walsh K., Schiaffino S., Lecker S.H., and Goldberg A.L. Foxo transcription factors induce the atrophy-related ubiquitin ligase atrogin-1 and cause skeletal muscle atrophy. Cell 117 (2004) 399-412
-
(2004)
Cell
, vol.117
, pp. 399-412
-
-
Sandri, M.1
Sandri, C.2
Gilbert, A.3
Skurk, C.4
Calabria, E.5
Picard, A.6
Walsh, K.7
Schiaffino, S.8
Lecker, S.H.9
Goldberg, A.L.10
-
23
-
-
0031010050
-
Regulation of skeletal muscle mass in mice by a new TGF-beta superfamily member
-
McPherron A.C., Lawler A.M., and Lee S.J. Regulation of skeletal muscle mass in mice by a new TGF-beta superfamily member. Nature 387 (1997) 83-90
-
(1997)
Nature
, vol.387
, pp. 83-90
-
-
McPherron, A.C.1
Lawler, A.M.2
Lee, S.J.3
-
24
-
-
33749254273
-
Myostatin induces cachexia by activating the ubiquitin proteolytic system through an NF-kappaB-independent, FoxO1-dependent mechanism
-
McFarlane C., Plummer E., Thomas M., Hennebry A., Ashby M., Ling N., Smith H., Sharma M., and Kambadur R. Myostatin induces cachexia by activating the ubiquitin proteolytic system through an NF-kappaB-independent, FoxO1-dependent mechanism. J. Cell Physiol. 209 (2006) 501-514
-
(2006)
J. Cell Physiol.
, vol.209
, pp. 501-514
-
-
McFarlane, C.1
Plummer, E.2
Thomas, M.3
Hennebry, A.4
Ashby, M.5
Ling, N.6
Smith, H.7
Sharma, M.8
Kambadur, R.9
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