Neuroprotective effects of leptin against ischemic injury induced by oxygen-glucose deprivation and transient cerebral ischemia

Stroke

Volumn 38, Issue 8, 2007, Pages 2329-2336

  Zhang, Feng ^a   Wang, Suping ^a   Signore, Armando P ^a   Chen, Jun ^a,b  

^a UNIVERSITY OF PITTSBURGH SCHOOL OF MEDICINE   (United States)

^b VETERANS AFFAIRS MEDICAL CENTER   (United States)

Author keywords

Brain ischemia; Extracellular signal related kinase 1 2; Leptin; Oxygen glucose deprivation

Indexed keywords

2 (2 AMINO 3 METHOXYPHENYL)CHROMONE; BRAIN RECEPTOR; GLUCOSE; LEPTIN RECEPTOR; MITOGEN ACTIVATED PROTEIN KINASE 1; MITOGEN ACTIVATED PROTEIN KINASE 3; OXYGEN; RECOMBINANT LEPTIN; CELL SURFACE RECEPTOR; LEPTIN; MITOGEN ACTIVATED PROTEIN KINASE; NEUROPROTECTIVE AGENT;

ANIMAL BEHAVIOR; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BODY WEIGHT; BRAIN CORTEX; BRAIN INFARCTION SIZE; BRAIN INJURY; BRAIN ISCHEMIA; BRAIN NERVE CELL; BRAIN PERFUSION; BRAIN PROTECTION; CONCENTRATION RESPONSE; CONTROLLED STUDY; CONVALESCENCE; CORPUS STRIATUM; DOSE RESPONSE; DRUG EFFECT; EMBRYO; ENZYME ACTIVATION; ENZYME PHOSPHORYLATION; HIPPOCAMPUS; IN VITRO STUDY; IN VIVO STUDY; MALE; MIDDLE CEREBRAL ARTERY OCCLUSION; NERVE CELL CULTURE; NERVE CELL NECROSIS; NEUROPROTECTION; NONHUMAN; PRIORITY JOURNAL; PROTEIN DETERMINATION; PROTEIN LOCALIZATION; RAT; REPERFUSION; TISSUE DISTRIBUTION; ANIMAL; APOPTOSIS; BRAIN; BRAIN INFARCTION; C57BL MOUSE; CELL CULTURE; CEREBRAL ARTERY DISEASE; DISEASE MODEL; DRUG POTENTIATION; METABOLISM; MOUSE; PATHOPHYSIOLOGY; PHOSPHORYLATION; PHYSIOLOGY; REPERFUSION INJURY; TRANSIENT ISCHEMIC ATTACK; TREATMENT OUTCOME; VASCULARIZATION;

ANIMALS; APOPTOSIS; BODY WEIGHT; BRAIN; BRAIN INFARCTION; CELLS, CULTURED; DISEASE MODELS, ANIMAL; EXTRACELLULAR SIGNAL-REGULATED MAP KINASES; GLUCOSE; INFARCTION, MIDDLE CEREBRAL ARTERY; ISCHEMIC ATTACK, TRANSIENT; LEPTIN; MALE; MICE; MICE, INBRED C57BL; NEUROPROTECTIVE AGENTS; PHOSPHORYLATION; RATS; RECEPTORS, CELL SURFACE; RECOVERY OF FUNCTION; REPERFUSION INJURY; TREATMENT OUTCOME;

EID: 34548256784     PISSN: 00392499     EISSN: None     Source Type: Journal    
DOI: 10.1161/STROKEAHA.107.482786     Document Type: Article

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