Apis mellifera venom and melittin block neither NF-κB-p50-DNA interactions nor the activation of NF-κB, instead they activate the transcription of proinflammatory genes and the release of reactive oxygen intermediates

Journal of Immunology

Volumn 179, Issue 1, 2007, Pages 655-664

  Stuhlmeier, Karl M ^a  

^a LUDWIG BOLTZMANN INSTITUTE FOR LUNG VASCULAR RESEARCH   (Austria)

Author keywords

[No Author keywords available]

Indexed keywords

BEE VENOM; CYCLOOXYGENASE 2; I KAPPA B; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; INTERLEUKIN 1BETA; MELITTIN; MESSENGER RNA; OXYGEN RADICAL; PROTEIN P50; REACTIVE OXYGEN METABOLITE;

ALTERNATIVE MEDICINE; ARTICLE; ASIA; CELL TYPE; CONTROLLED STUDY; DRUG EFFICACY; DRUG EXPOSURE; GEL MOBILITY SHIFT ASSAY; HUMAN; HUMAN CELL; IMMUNOMODULATION; INFLAMMATION; LEUKOCYTE; MARKER GENE; MONONUCLEAR CELL; PRIORITY JOURNAL; PROTEIN DEGRADATION; PROTEIN DNA BINDING; PROTEIN PHOSPHORYLATION; PROTEIN PROTEIN INTERACTION; RHEUMATOID ARTHRITIS; SKIN FIBROBLAST; SYNOVIOCYTE; TRANSCRIPTION INITIATION; WESTERN BLOTTING; WESTERN HEMISPHERE;

EID: 34250861997     PISSN: 00221767     EISSN: None     Source Type: Journal    
DOI: 10.4049/jimmunol.179.1.655     Document Type: Article

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