F2L, a peptide derived from heme-binding protein, inhibits formyl peptide receptor-mediated signaling

Biochemical and Biophysical Research Communications

Volumn 359, Issue 4, 2007, Pages 985-990

  Lee, Ha Young ^a   Lee, Sun Young ^a   Shin, Eun Ha ^a   Kim, Sang Doo ^a   Kim, Jung Mo ^a   Lee, Mi Sook ^b   Ryu, Sung Ho ^b   Bae, Yoe Sik ^a  

^a Dong A University   (South Korea)

^b Pohang University of Science and Technology (POSTECH)   (South Korea)

Author keywords

F2L; fMLF; Formyl peptide receptor; Neutrophil; Superoxide generation; WKYMVm

Indexed keywords

CALCIUM; CHEMOTACTIC FACTOR; FORMYLPEPTIDE RECEPTOR; HEMOPROTEIN; IODINE 125; LEUCINE; LYSINE; METHIONINE; PHENYLALANINE; PROTEIN F2L; PROTEIN MMK 1; SUPEROXIDE; TRYPTOPHAN; TYROSINE; UNCLASSIFIED DRUG; VALINE; VEGETABLE PROTEIN;

ARTICLE; CELL MIGRATION; CELL STIMULATION; CONTROLLED STUDY; DRUG INHIBITION; HUMAN; HUMAN CELL; NEUTROPHIL; PRIORITY JOURNAL; PROTEIN BINDING; SIGNAL TRANSDUCTION;

CARRIER PROTEINS; CELLS, CULTURED; CHEMOTACTIC FACTORS; CHEMOTAXIS; DOSE-RESPONSE RELATIONSHIP, DRUG; HEMEPROTEINS; HUMANS; NEUTROPHILS; PEPTIDES; RECEPTORS, FORMYL PEPTIDE; RECEPTORS, LIPOXIN;

EID: 34250634444     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2007.06.001     Document Type: Article

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