A defect in DNA double strand break processing in cells from unaffected parents of retinoblastoma patients and other apparently normal humans

DNA Repair

Volumn 6, Issue 6, 2007, Pages 818-829

  Kato, Takamitsu A ^a   Wilson, Paul F ^a,d   Nagasawa, Hatsumi ^a   Fitzek, Markus M ^b   Weil, Michael M ^a   Little, John B ^c   Bedford, Joel S ^a  

^a Department of Environmental and Radiological Health Sciences   (United States)

^b TUFTS MEDICAL CENTER   (United States)

^c HARVARD SCHOOL OF PUBLIC HEALTH   (United States)

^d DNA Repair Group   (United States)

Author keywords

DNA DSB; Gamma radiation; Gamma H2AX; Retinoblastoma

Indexed keywords

DOUBLE STRANDED DNA; HISTONE H2AX;

ARTICLE; CELL CYCLE ARREST; CELL CYCLE G0 PHASE; CELL CYCLE G2 PHASE; CELL CYCLE M PHASE; CELL DAMAGE; CELL KILLING; CELL STRAIN; CLINICAL ARTICLE; CONTROLLED STUDY; DNA STRAND BREAKAGE; GAMMA RADIATION; HUMAN; HUMAN CELL; LOW ENERGY RADIATION; PRIORITY JOURNAL; RADIOSENSITIVITY; RETINOBLASTOMA;

CELL CYCLE; CELL DIVISION; DNA BREAKS, DOUBLE-STRANDED; DNA DAMAGE; DNA REPAIR; FAMILY HEALTH; G0 PHASE; G2 PHASE; GERM-LINE MUTATION; HISTONES; HUMANS; IMMUNOHISTOCHEMISTRY; RECOMBINATION, GENETIC; RETINOBLASTOMA; TIME FACTORS;

EID: 34247582841     PISSN: 15687864     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.dnarep.2007.01.008     Document Type: Article

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