-
1
-
-
0030805991
-
Frequency of stages of Alzheimer-related lesions in different age categories
-
Braak H, Braak E. Frequency of stages of Alzheimer-related lesions in different age categories. Neurobiol Aging. 1997;18:351-357.
-
(1997)
Neurobiol Aging
, vol.18
, pp. 351-357
-
-
Braak, H.1
Braak, E.2
-
2
-
-
0026512066
-
Identification of normal and pathological aging in prospectively studied nondemented elderly humans
-
Dickson DW, Crystal HA, Mattiace LA, et al. Identification of normal and pathological aging in prospectively studied nondemented elderly humans. Neurobiol Aging. 1992;13:179-189.
-
(1992)
Neurobiol Aging
, vol.13
, pp. 179-189
-
-
Dickson, D.W.1
Crystal, H.A.2
Mattiace, L.A.3
-
3
-
-
0028174629
-
β Amyloid protein deposition in the brain after severe head injury: Implications for the pathogenesis of Alzheimer's disease
-
Roberts GW, Gentleman SM, Lynch A, et al. β Amyloid protein deposition in the brain after severe head injury: implications for the pathogenesis of Alzheimer's disease. J Neurol Neurosurg Psychiatry. 1994;57:419-425.
-
(1994)
J Neurol Neurosurg Psychiatry
, vol.57
, pp. 419-425
-
-
Roberts, G.W.1
Gentleman, S.M.2
Lynch, A.3
-
4
-
-
0036927213
-
Changes in expression of amyloid precursor protein and interleukin-1β after experimental traumatic brain injury in rats
-
Ciallella JR, Ikonomovic MD, Paljug WR, et al. Changes in expression of amyloid precursor protein and interleukin-1β after experimental traumatic brain injury in rats. J Neurotrauma. 2002;19:1555-1567.
-
(2002)
J Neurotrauma
, vol.19
, pp. 1555-1567
-
-
Ciallella, J.R.1
Ikonomovic, M.D.2
Paljug, W.R.3
-
5
-
-
0031770501
-
Experimental brain injury induces expression of amyloid precursor protein, which may be related to neuronal loss in the hippocampus
-
Murakami N, Yamaki T, Iwamoto Y, et al. Experimental brain injury induces expression of amyloid precursor protein, which may be related to neuronal loss in the hippocampus. J Neurotrauma. 1998;15:993-1003.
-
(1998)
J Neurotrauma
, vol.15
, pp. 993-1003
-
-
Murakami, N.1
Yamaki, T.2
Iwamoto, Y.3
-
6
-
-
0027482350
-
β-Amyloid precursor protein (βAPP) as a marker for axonal injury after head injury
-
Gentleman SM, Nash MJ, Sweeting CJ, et al. β-Amyloid precursor protein (βAPP) as a marker for axonal injury after head injury. Neurosci Lett. 1993;160:139-144.
-
(1993)
Neurosci Lett
, vol.160
, pp. 139-144
-
-
Gentleman, S.M.1
Nash, M.J.2
Sweeting, C.J.3
-
7
-
-
0037404532
-
Amyloid beta accumulation in axons after traumatic brain injury in humans
-
Smith DH, Chen XH, Iwata A, Graham DI. Amyloid beta accumulation in axons after traumatic brain injury in humans. J Neurosurg. 2003;98:1072-1077.
-
(2003)
J Neurosurg
, vol.98
, pp. 1072-1077
-
-
Smith, D.H.1
Chen, X.H.2
Iwata, A.3
Graham, D.I.4
-
8
-
-
5044237263
-
Alzheimer's pathology in human temporal cortex surgically excised after severe brain injury
-
Ikonomovic MD, Uryu K, Abrahamson EE, et al. Alzheimer's pathology in human temporal cortex surgically excised after severe brain injury. Exp Neurol. 2004;190:192-203.
-
(2004)
Exp Neurol
, vol.190
, pp. 192-203
-
-
Ikonomovic, M.D.1
Uryu, K.2
Abrahamson, E.E.3
-
9
-
-
0142120762
-
Alterations in cerebrospinal fluid apolipoprotein E and amyloid β-protein after traumatic brain injury
-
Kay AD, Petzold A, Kerr M, et al. Alterations in cerebrospinal fluid apolipoprotein E and amyloid β-protein after traumatic brain injury. J Neurotrauma. 2003;20:943-952.
-
(2003)
J Neurotrauma
, vol.20
, pp. 943-952
-
-
Kay, A.D.1
Petzold, A.2
Kerr, M.3
-
10
-
-
0038117796
-
Correlation between elevated levels of amyloid β-peptide in the brain and cognitive decline
-
Naslund J, Haroutunian V, Mohs R, et al. Correlation between elevated levels of amyloid β-peptide in the brain and cognitive decline. JAMA. 2000;283:1571-1577.
-
(2000)
JAMA
, vol.283
, pp. 1571-1577
-
-
Naslund, J.1
Haroutunian, V.2
Mohs, R.3
-
11
-
-
5344277556
-
Deciphering the molecular basis of memory failure in Alzheimer's disease
-
Walsh DM, Selkoe DJ. Deciphering the molecular basis of memory failure in Alzheimer's disease. Neuron. 2004;44:181-193.
-
(2004)
Neuron
, vol.44
, pp. 181-193
-
-
Walsh, D.M.1
Selkoe, D.J.2
-
12
-
-
33745268224
-
Different conformations of amyloid β induce neurotoxicity by distinct mechanisms in human cortical neurons
-
Deshpande A, Mina E, Glabe C, Busciglio J. Different conformations of amyloid β induce neurotoxicity by distinct mechanisms in human cortical neurons. J Neurosci. 2006;26:6011-6018.
-
(2006)
J Neurosci
, vol.26
, pp. 6011-6018
-
-
Deshpande, A.1
Mina, E.2
Glabe, C.3
Busciglio, J.4
-
13
-
-
33646364811
-
Cerebrolysin decreases amyloid-β production by regulating amyloid protein precursor maturation in a transgenic model of Alzheimer's disease
-
Rockenstein E, Torrance M, Mante M, et al. Cerebrolysin decreases amyloid-β production by regulating amyloid protein precursor maturation in a transgenic model of Alzheimer's disease. J Neurosci Res. 2006;83:1252-1261.
-
(2006)
J Neurosci Res
, vol.83
, pp. 1252-1261
-
-
Rockenstein, E.1
Torrance, M.2
Mante, M.3
-
14
-
-
33646938005
-
Short amyloid-beta (Aβ) immunogens reduce cerebral Aβ load and learning deficits in an Alzheimer's disease mouse model in the absence of an Aβ-specific cellular immune response
-
Maier M, Seabrook TJ, Lazo ND, et al. Short amyloid-beta (Aβ) immunogens reduce cerebral Aβ load and learning deficits in an Alzheimer's disease mouse model in the absence of an Aβ-specific cellular immune response. J Neurosci. 2006;26:4717-4728.
-
(2006)
J Neurosci
, vol.26
, pp. 4717-4728
-
-
Maier, M.1
Seabrook, T.J.2
Lazo, N.D.3
-
15
-
-
0032837741
-
The levels of soluble versus insoluble brain Aβ distinguish Alzheimer's disease from normal and pathologic aging
-
Wang J, Dickson DW, Trojanowski JQ, Lee VM. The levels of soluble versus insoluble brain Aβ distinguish Alzheimer's disease from normal and pathologic aging. Exp Neurol. 1999;158:328-337.
-
(1999)
Exp Neurol
, vol.158
, pp. 328-337
-
-
Wang, J.1
Dickson, D.W.2
Trojanowski, J.Q.3
Lee, V.M.4
-
16
-
-
0030612292
-
Aβ42 is the predominant form of amyloid β-protein in the brains of short-term survivors of head injury
-
Gentleman SM, Greenberg BD, Savage MJ, et al. Aβ42 is the predominant form of amyloid β-protein in the brains of short-term survivors of head injury. Neuroreport. 1997;8:1519-1522.
-
(1997)
Neuroreport
, vol.8
, pp. 1519-1522
-
-
Gentleman, S.M.1
Greenberg, B.D.2
Savage, M.J.3
-
17
-
-
0033625502
-
β-Amyloid (Aβ)42(43), aβ42, aβ40 and apoE immunostaining of plaques in fatal head injury
-
Horsburgh K, Cole GM, Yang F, et al. β-Amyloid (Aβ)42(43), aβ42, aβ40 and apoE immunostaining of plaques in fatal head injury. Neuropathol Appl Neurobiol. 2000;26:124-132.
-
(2000)
Neuropathol Appl Neurobiol
, vol.26
, pp. 124-132
-
-
Horsburgh, K.1
Cole, G.M.2
Yang, F.3
-
18
-
-
0028169925
-
Visualization of Aβ42(43) and Aβ40 in senile plaques with end-specific Aβ monoclonals: Evidence that an initially deposited species is Aβ42(43)
-
Iwatsubo T, Odaka A, Suzuki N, et al. Visualization of Aβ42(43) and Aβ40 in senile plaques with end-specific Aβ monoclonals: evidence that an initially deposited species is Aβ42(43). Neuron. 1994;13:45-53.
-
(1994)
Neuron
, vol.13
, pp. 45-53
-
-
Iwatsubo, T.1
Odaka, A.2
Suzuki, N.3
-
19
-
-
0028670780
-
Increased numbers of βAPP-immunoreactive neurones in the entorhinal cortex after head injury
-
McKenzie JE, Gentleman SM, Roberts GW, et al. Increased numbers of βAPP-immunoreactive neurones in the entorhinal cortex after head injury. Neuroreport. 1994;6:161-164.
-
(1994)
Neuroreport
, vol.6
, pp. 161-164
-
-
McKenzie, J.E.1
Gentleman, S.M.2
Roberts, G.W.3
-
20
-
-
33644832047
-
Inverse relation between in vivo amyloid imaging load and cerebrospinal fluid Aβ42 in humans
-
Fagan AM, Mintun MA, Mach RH, et al. Inverse relation between in vivo amyloid imaging load and cerebrospinal fluid Aβ42 in humans. Ann Neurol. 2006;59:512-519.
-
(2006)
Ann Neurol
, vol.59
, pp. 512-519
-
-
Fagan, A.M.1
Mintun, M.A.2
Mach, R.H.3
-
21
-
-
0028986690
-
Apolipoprotein E epsilon 4 allele is associated with deposition of amyloid β-protein following head injury
-
Nicoll JA, Roberts GW, Graham DI. Apolipoprotein E epsilon 4 allele is associated with deposition of amyloid β-protein following head injury. Nat Med. 1995;1:135-137.
-
(1995)
Nat Med
, vol.1
, pp. 135-137
-
-
Nicoll, J.A.1
Roberts, G.W.2
Graham, D.I.3
-
22
-
-
0029018224
-
Synergistic effects of traumatic head injury and apolipoprotein-E4 in patients with Alzheimer's disease
-
Mayeux R, Ottman R, Maestre G, et al. Synergistic effects of traumatic head injury and apolipoprotein-E4 in patients with Alzheimer's disease. Neurology. 1995;45:555-557.
-
(1995)
Neurology
, vol.45
, pp. 555-557
-
-
Mayeux, R.1
Ottman, R.2
Maestre, G.3
-
23
-
-
0028808165
-
Are the pathobiological changes evoked by traumatic brain injury immediate and irreversible?
-
Povlishock JT, Jenkins LW. Are the pathobiological changes evoked by traumatic brain injury immediate and irreversible? Brain Pathol. 1995;5:415-426.
-
(1995)
Brain Pathol
, vol.5
, pp. 415-426
-
-
Povlishock, J.T.1
Jenkins, L.W.2
-
24
-
-
0026570528
-
β-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicity
-
Mattson MP, Cheng B, Davis D, et al. β-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicity. J Neurosci. 1992;12:376-389.
-
(1992)
J Neurosci
, vol.12
, pp. 376-389
-
-
Mattson, M.P.1
Cheng, B.2
Davis, D.3
-
25
-
-
33645038471
-
A specific amyloid-β protein assembly in the brain impairs memory
-
Lesne S, Koh MT, Kotilinek L, et al. A specific amyloid-β protein assembly in the brain impairs memory. Nature. 2006;440:352-357.
-
(2006)
Nature
, vol.440
, pp. 352-357
-
-
Lesne, S.1
Koh, M.T.2
Kotilinek, L.3
-
26
-
-
16644379264
-
Natural oligomers of the amyloid-β protein specifically disrupt cognitive function
-
Cleary JP, Walsh DM, Hofmeister JJ, et al. Natural oligomers of the amyloid-β protein specifically disrupt cognitive function. Nat Neurosci. 2005;8:79-84.
-
(2005)
Nat Neurosci
, vol.8
, pp. 79-84
-
-
Cleary, J.P.1
Walsh, D.M.2
Hofmeister, J.J.3
|