Altered susceptibility to EAE in congenic NOD mice: Altered processing of the encephalitogenic MOG35-55 peptide by NOR/LtJ mice

Clinical Immunology

Volumn 122, Issue 1, 2007, Pages 91-100

  Mayo, Stella ^a   Quinn, Anthony ^a  

^a UNIVERSITY OF TOLEDO   (United States)

Author keywords

Antigen processing; CTL; Determinants; EAE; Epitopes; MHC; MOG; NOD; NOR

Indexed keywords

ENCEPHALITOGENIC PROTEIN; MYELIN PROTEIN; MYELIN PROTEIN [35-55]; UNCLASSIFIED DRUG;

ALLERGIC ENCEPHALOMYELITIS; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; AUTOIMMUNE DISEASE; COMPARATIVE STUDY; CONTROLLED STUDY; CYTOTOXIC T LYMPHOCYTE; DISEASE SEVERITY; GENETIC SUSCEPTIBILITY; IMMUNE RESPONSE; IMMUNIZATION; IMMUNOREGULATION; LYMPHOBLAST; MOUSE; MOUSE STRAIN; NONHUMAN; PRIORITY JOURNAL; PROTEIN PROCESSING; T LYMPHOCYTE;

ANIMALS; CD8-POSITIVE T-LYMPHOCYTES; CYTOKINES; ENCEPHALOMYELITIS, AUTOIMMUNE, EXPERIMENTAL; ENZYME-LINKED IMMUNOSORBENT ASSAY; EPITOPES, T-LYMPHOCYTE; FLOW CYTOMETRY; GENETIC PREDISPOSITION TO DISEASE; GLYCOPROTEINS; MAJOR HISTOCOMPATIBILITY COMPLEX; MICE; MICE, CONGENIC; MICE, INBRED NOD; PEPTIDE FRAGMENTS;

EID: 33845878795     PISSN: 15216616     EISSN: 15217035     Source Type: Journal    
DOI: 10.1016/j.clim.2006.08.001     Document Type: Article

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