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Volumn 71, Issue 4, 2006, Pages 612-614

iNOS - Another cardiac target of calcineurin

Author keywords

[No Author keywords available]

Indexed keywords

CALCINEURIN; CYCLOSPORIN A; INDUCIBLE NITRIC OXIDE SYNTHASE; TRANSCRIPTION FACTOR NFAT;

EID: 33751577204     PISSN: 00086363     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.cardiores.2006.07.007     Document Type: Editorial
Times cited : (5)

References (23)
  • 1
    • 0001018554 scopus 로고
    • Calcineurin: a calcium- and calmodulin-binding protein of the nervous system
    • Klee C.B., Crouch T.H., and Krinks M.H. Calcineurin: a calcium- and calmodulin-binding protein of the nervous system. Proc Natl Acad Sci U S A 76 (1979) 6270-6273
    • (1979) Proc Natl Acad Sci U S A , vol.76 , pp. 6270-6273
    • Klee, C.B.1    Crouch, T.H.2    Krinks, M.H.3
  • 2
    • 0032540267 scopus 로고    scopus 로고
    • A calcineurin-dependent transcriptional pathway for cardiac hypertrophy
    • Molkentin J.D., Lu J.R., Antos C.L., Markham B., Richardson J., Robbins J., et al. A calcineurin-dependent transcriptional pathway for cardiac hypertrophy. Cell 93 (1998) 215-228
    • (1998) Cell , vol.93 , pp. 215-228
    • Molkentin, J.D.1    Lu, J.R.2    Antos, C.L.3    Markham, B.4    Richardson, J.5    Robbins, J.6
  • 3
    • 33751577221 scopus 로고    scopus 로고
    • Calcineurin regulates NFAT-dependent iNOS expression and protection of cardiomyocytes: co-operation with Src tyrosine kinase
    • Obasanji-Blackshire K., Mesquita R., Molkentin J.D., Hart S.L., Marber M.S., Xia Y., et al. Calcineurin regulates NFAT-dependent iNOS expression and protection of cardiomyocytes: co-operation with Src tyrosine kinase. Cardiovasc Res 71 (2006) 672-683
    • (2006) Cardiovasc Res , vol.71 , pp. 672-683
    • Obasanji-Blackshire, K.1    Mesquita, R.2    Molkentin, J.D.3    Hart, S.L.4    Marber, M.S.5    Xia, Y.6
  • 4
    • 0347320747 scopus 로고    scopus 로고
    • Calcineurin/NFAT coupling participates in pathological, but not physiological, cardiac hypertrophy
    • Wilkins B.J., Dai Y.S., Bueno O.F., Parsons S.A., Xu J., Plank D.M., et al. Calcineurin/NFAT coupling participates in pathological, but not physiological, cardiac hypertrophy. Circ Res 94 (2004) 110-118
    • (2004) Circ Res , vol.94 , pp. 110-118
    • Wilkins, B.J.1    Dai, Y.S.2    Bueno, O.F.3    Parsons, S.A.4    Xu, J.5    Plank, D.M.6
  • 5
    • 2942746423 scopus 로고    scopus 로고
    • Genetic loss of calcineurin blocks mechanical overload-induced skeletal muscle fiber type switching but not hypertrophy
    • Parsons S.A., Millay D.P., Wilkins B.J., Bueno O.F., Tsika G.L., and Neilson J.R. Genetic loss of calcineurin blocks mechanical overload-induced skeletal muscle fiber type switching but not hypertrophy. J Biol Chem 279 (2004) 26192-26200
    • (2004) J Biol Chem , vol.279 , pp. 26192-26200
    • Parsons, S.A.1    Millay, D.P.2    Wilkins, B.J.3    Bueno, O.F.4    Tsika, G.L.5    Neilson, J.R.6
  • 6
    • 27644437324 scopus 로고    scopus 로고
    • PKC-independent signal transduction pathways increase SERCA2 expression in adult rat cardiomyocytes
    • Anwar A., Taimor G., Korkususz H., Schreckenberg R., Berndt T., Abdallah Y., et al. PKC-independent signal transduction pathways increase SERCA2 expression in adult rat cardiomyocytes. J Mol Cell Cardiol 39 (2005) 911-919
    • (2005) J Mol Cell Cardiol , vol.39 , pp. 911-919
    • Anwar, A.1    Taimor, G.2    Korkususz, H.3    Schreckenberg, R.4    Berndt, T.5    Abdallah, Y.6
  • 7
    • 0034705070 scopus 로고    scopus 로고
    • Calcineurin expression, activation, and function in cardiac pressure-overload hypertrophy
    • Lim H.W., De Windt L.J., Steinberg L., Taigen T., Witt S.A., Kimball T.R., et al. Calcineurin expression, activation, and function in cardiac pressure-overload hypertrophy. Circulation 101 (2000) 2431-2437
    • (2000) Circulation , vol.101 , pp. 2431-2437
    • Lim, H.W.1    De Windt, L.J.2    Steinberg, L.3    Taigen, T.4    Witt, S.A.5    Kimball, T.R.6
  • 8
    • 0034691296 scopus 로고    scopus 로고
    • Cardiac hypertrophy is not a required compensatory response to short-term pressure overload
    • Hill J.A., Karimi M., Kutschke W., Davisson R.L., Zimmerman K., Wang Z., et al. Cardiac hypertrophy is not a required compensatory response to short-term pressure overload. Circulation 101 (2000) 2863-2869
    • (2000) Circulation , vol.101 , pp. 2863-2869
    • Hill, J.A.1    Karimi, M.2    Kutschke, W.3    Davisson, R.L.4    Zimmerman, K.5    Wang, Z.6
  • 9
    • 0033515619 scopus 로고    scopus 로고
    • Pressure overload induces severe hypertrophy in mice treated with cyclosporine, an inhibitor of calcineurin
    • Ding B., Price R.L., Borg T.K., Weinberg E.O., Halloran P.F., Lorell B.H., et al. Pressure overload induces severe hypertrophy in mice treated with cyclosporine, an inhibitor of calcineurin. Circ Res 84 (1999) 729-734
    • (1999) Circ Res , vol.84 , pp. 729-734
    • Ding, B.1    Price, R.L.2    Borg, T.K.3    Weinberg, E.O.4    Halloran, P.F.5    Lorell, B.H.6
  • 10
    • 0037453589 scopus 로고    scopus 로고
    • NFAT transcription factors are critical survival factors that inhibit cardiomyocyte apoptosis during phenylephrine stimulation in vitro
    • Pu W.T., Ma Q., and Izumo S. NFAT transcription factors are critical survival factors that inhibit cardiomyocyte apoptosis during phenylephrine stimulation in vitro. Circ Res 92 (2003) 725-731
    • (2003) Circ Res , vol.92 , pp. 725-731
    • Pu, W.T.1    Ma, Q.2    Izumo, S.3
  • 11
    • 0035512186 scopus 로고    scopus 로고
    • Transcription activator protein 1 (AP-1) mediates NO-induced apoptosis of adult cardiomyocytes
    • Taimor G., Rakow A., and Piper H.M. Transcription activator protein 1 (AP-1) mediates NO-induced apoptosis of adult cardiomyocytes. FASEB J 15 (2001) 2518-2520
    • (2001) FASEB J , vol.15 , pp. 2518-2520
    • Taimor, G.1    Rakow, A.2    Piper, H.M.3
  • 12
    • 0035199811 scopus 로고    scopus 로고
    • Cardioprotective function of inducible nitric oxide synthase and role of nitric oxide in myocardial ischemia and preconditioning: an overview of a decade of research
    • Bolli R. Cardioprotective function of inducible nitric oxide synthase and role of nitric oxide in myocardial ischemia and preconditioning: an overview of a decade of research. J Mol Cell Cardiol 33 (2001) 1897-1918
    • (2001) J Mol Cell Cardiol , vol.33 , pp. 1897-1918
    • Bolli, R.1
  • 13
  • 14
    • 0037059465 scopus 로고    scopus 로고
    • Cardiac-specific overexpression of inducible nitric oxide synthase does not result in severe cardiac dysfunction
    • Heger J., Gödecke A., Flögel U., Merx M.W., Molojavyi A., Kühn-Velten W.N., et al. Cardiac-specific overexpression of inducible nitric oxide synthase does not result in severe cardiac dysfunction. Circ Res 90 (2002) 93-99
    • (2002) Circ Res , vol.90 , pp. 93-99
    • Heger, J.1    Gödecke, A.2    Flögel, U.3    Merx, M.W.4    Molojavyi, A.5    Kühn-Velten, W.N.6
  • 15
    • 0038498077 scopus 로고    scopus 로고
    • Myoglobin protects the heart from inducible nitric-oxide synthase (iNOS)-mediated nitrosative stress
    • Gödecke A., Molojavyi A., Heger J., Flogel U., Ding Z., Jacoby C., et al. Myoglobin protects the heart from inducible nitric-oxide synthase (iNOS)-mediated nitrosative stress. J Biol Chem 278 (2003) 21761-21776
    • (2003) J Biol Chem , vol.278 , pp. 21761-21776
    • Gödecke, A.1    Molojavyi, A.2    Heger, J.3    Flogel, U.4    Ding, Z.5    Jacoby, C.6
  • 16
    • 0033597755 scopus 로고    scopus 로고
    • Myocardial ischemia/reperfusion injury in the inducible nitric oxide synthase knockout mice
    • Xi L., Jarrett N.C., Hess M.L., and Kukreja R.C. Myocardial ischemia/reperfusion injury in the inducible nitric oxide synthase knockout mice. Life Sci 65 (1999) 935-945
    • (1999) Life Sci , vol.65 , pp. 935-945
    • Xi, L.1    Jarrett, N.C.2    Hess, M.L.3    Kukreja, R.C.4
  • 17
    • 0037453583 scopus 로고    scopus 로고
    • Gene therapy with inducible nitric oxide synthase protects against myocardial infarction via a cyclooxygenase-2-dependent mechanism
    • Li Q., Guo Y., Xuan Y.T., Lowenstein C.J., Stevenson S.C., Prabhu S.D., et al. Gene therapy with inducible nitric oxide synthase protects against myocardial infarction via a cyclooxygenase-2-dependent mechanism. Circ Res 92 (2003) 741-748
    • (2003) Circ Res , vol.92 , pp. 741-748
    • Li, Q.1    Guo, Y.2    Xuan, Y.T.3    Lowenstein, C.J.4    Stevenson, S.C.5    Prabhu, S.D.6
  • 18
    • 33644871719 scopus 로고    scopus 로고
    • Gene therapy with iNOS provides long-term protection against myocardial infarction without adverse functional consequences
    • Li Q., Guo Y., Tan W., Stein A.B., Dawn B., Wu W.J., et al. Gene therapy with iNOS provides long-term protection against myocardial infarction without adverse functional consequences. Am J Physiol Heart Circ Physiol 290 (2006) H584-H589
    • (2006) Am J Physiol Heart Circ Physiol , vol.290
    • Li, Q.1    Guo, Y.2    Tan, W.3    Stein, A.B.4    Dawn, B.5    Wu, W.J.6
  • 19
    • 29444432915 scopus 로고    scopus 로고
    • The ubiquitous role of nitric oxide in cardioprotection
    • Jones S.P., and Bolli R. The ubiquitous role of nitric oxide in cardioprotection. J Mol Cell Cardiol 40 (2006) 16-23
    • (2006) J Mol Cell Cardiol , vol.40 , pp. 16-23
    • Jones, S.P.1    Bolli, R.2
  • 20
    • 0033566593 scopus 로고    scopus 로고
    • Inducible nitric oxide synthase activation after ischemia/reperfusion contributes to myocardial dysfunction and extent of infarct size in rabbits: evidence for a late phase of nitric oxide-mediated reperfusion injury
    • Wildhirt S.M., Weismueller S., Schulze C., Conrad N., Kornberg A., and Reichart B. Inducible nitric oxide synthase activation after ischemia/reperfusion contributes to myocardial dysfunction and extent of infarct size in rabbits: evidence for a late phase of nitric oxide-mediated reperfusion injury. Cardiovasc Res 43 (1999) 698-711
    • (1999) Cardiovasc Res , vol.43 , pp. 698-711
    • Wildhirt, S.M.1    Weismueller, S.2    Schulze, C.3    Conrad, N.4    Kornberg, A.5    Reichart, B.6
  • 21
    • 0036268389 scopus 로고    scopus 로고
    • Role of nitric oxide in myocardial preconditioning
    • Dawn B., and Bolli R. Role of nitric oxide in myocardial preconditioning. Ann N Y Acad Sci 962 (2002) 18-41
    • (2002) Ann N Y Acad Sci , vol.962 , pp. 18-41
    • Dawn, B.1    Bolli, R.2
  • 22
    • 1542317022 scopus 로고    scopus 로고
    • Ca2+/calmodulin-dependent protein phosphatase calcineurin mediates the expression of iNOS through IKK and NF-kappaB activity in LPS-stimulated mouse peritoneal macrophages and RAW 264.7 cells
    • Kim Y., Moon J.S., Lee K.S., Park S.Y., Cheong J., Kang H.S., et al. Ca2+/calmodulin-dependent protein phosphatase calcineurin mediates the expression of iNOS through IKK and NF-kappaB activity in LPS-stimulated mouse peritoneal macrophages and RAW 264.7 cells. Biochem Biophys Res Commun 314 (2004) 695-703
    • (2004) Biochem Biophys Res Commun , vol.314 , pp. 695-703
    • Kim, Y.1    Moon, J.S.2    Lee, K.S.3    Park, S.Y.4    Cheong, J.5    Kang, H.S.6
  • 23
    • 33746848336 scopus 로고    scopus 로고
    • Knockdown of NFAT3 blocked TPA-induced COX-2 and iNOS expression, and enhanced cell transformation in Cl41 cells
    • 10.1002/jcb.20834 [Electronic publication ahead of print]
    • Li J., Song L., Zhang D., Wei L., and Huang C. Knockdown of NFAT3 blocked TPA-induced COX-2 and iNOS expression, and enhanced cell transformation in Cl41 cells. J Cell Biochem (2006) 10.1002/jcb.20834 [Electronic publication ahead of print]
    • (2006) J Cell Biochem
    • Li, J.1    Song, L.2    Zhang, D.3    Wei, L.4    Huang, C.5


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.