Gene-environment factors that contribute to alcoholic pancreatitis in humans

Journal of Gastroenterology and Hepatology (Australia)

Volumn 21, Issue SUPPL. 3, 2006, Pages

  Whitcomb, David C ^a,b  

^a UNIVERSITY OF PITTSBURGH MEDICAL CENTER   (United States)

^b PRESBYTERIAN UNIVERSITY HOSPITAL   (United States)

Author keywords

Cerulean; Cystic fibrosis transmembrane conductance inhibitor (CFTR); Fibrosis; Human; Pancreatic secretory trypsin inhibitor (SPINK1); Rat; Stellate cell; Trypsin; Trypsinogen

Indexed keywords

ADENOSINE TRIPHOSPHATE; ALCOHOL; APROTININ; BETHANECHOL; CALCIUM; CHOLECYSTOKININ; COLLAGEN TYPE 1; DEOXYGLUCOSE; FATTY ACID ESTER; PROCOLLAGEN; TRANSFORMING GROWTH FACTOR BETA; TRANSMEMBRANE CONDUCTANCE REGULATOR; TRYPSIN; TRYPSIN INHIBITOR; TRYPSINOGEN;

ACUTE PANCREATITIS; ALCOHOL CONSUMPTION; ALCOHOL METABOLISM; ALCOHOLIC PANCREATITIS; AREA POSTREMA; CHRONIC PANCREATITIS; CONFERENCE PAPER; CYSTIC FIBROSIS; CYTOKINE RELEASE; ENVIRONMENTAL FACTOR; GENE; GENE MUTATION; GENETIC EPIDEMIOLOGY; GENETIC SUSCEPTIBILITY; GENOTYPE ENVIRONMENT INTERACTION; HAPLOTYPE; HEREDITY; HUMAN; PRIORITY JOURNAL; PRSS1 GENE; SMOKING; SPINK1 GENE; SYSTEMATIC REVIEW;

EID: 33747599858     PISSN: 08159319     EISSN: 14401746     Source Type: Journal    
DOI: 10.1111/j.1440-1746.2006.04583.x     Document Type: Conference Paper

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