Ablation of gap junctional communication in hepatocytes of transgenic mice does not lead to disrupted cellular homeostasis or increased spontaneous tumourigenesis

European Journal of Cell Biology

Volumn 85, Issue 8, 2006, Pages 717-728

  Ott, Thomas ^a   Jokwitz, Melanie ^a   Lenhard, Diana ^a   Romualdi, Alessandro ^b   Dombrowski, Frank ^c   Ittrich, Carina ^d   Schwarz, Michael ^e   Willecke, Klaus ^a  

^a UNIVERSITY OF BONN   (Germany)

^b FRITZ LIPMANN INSTITUTE   (Germany)

^c OTTO VON GUERICKE UNIVERSITY   (Germany)

^d GERMAN CANCER RESEARCH CENTER DKFZ   (Germany)

^e UNIVERSITY OF TÜBINGEN   (Germany)

Author keywords

Connexin26; Connexin32; Cx26, Connexin26; Cx32, Connexin32; db cAMP, Dibutyryl 3,5 cyclic adenosine monophosphate; Gap junctions; Gene targeting; GJIC, Gap junctional intercellular communication; Liver; PH, Partial hepatectomy; Proliferation

Indexed keywords

ACTIN; CONNEXIN 26; CONNEXIN 32; CRE RECOMBINASE; DNA; PROTEIN SUBUNIT; UVOMORULIN;

AMYLOIDOSIS; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CANCER INHIBITION; CELL COMMUNICATION; CELL FUNCTION; CELL PROLIFERATION; CELL TYPE; CHEMICAL CARCINOGENESIS; CONTROLLED STUDY; CROSSING OVER; FERTILITY; GAP JUNCTION; GENE TARGETING; HOMEOSTASIS; LIVER CARCINOGENESIS; LIVER CELL; LOXP SITE; MICROARRAY ANALYSIS; MOUSE; NONHUMAN; NUCLEOTIDE SEQUENCE; PHENOTYPE; PRECANCER; PRIORITY JOURNAL; PROGENY; PROTEIN DEFICIENCY; PROTEIN EXPRESSION; TRANSGENIC MOUSE;

MURINAE; MUS MUSCULUS;

EID: 33746047519     PISSN: 01719335     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.ejcb.2006.03.004     Document Type: Article

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